PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2836539-6	1988	DiC8 inhibited the rise in [Ca2+]i elicited with FMLP, LTB4, and AGEPC; this effect, as well as the DG-mediated suppression of degranulation, could be reversed with the protein kinase C (PKC) inhibitor, 1-(-5-isoquinolinesulfonyl)-2-methylpiperazine hydrochloride (H-7).	1,2-dioctanoylglycerol	0-4	formyl peptide receptor 1	Homo sapiens	49-53	
2174064-9	1990	Third, following neutrophil priming with dioctanoylglycerol (diC8), maximal O2- production occurred in response to 0.015 microM fMLP or Con A without a rise in [Ca]i, and diC8/fMLP-induced O2- production was not inhibited by EGTA.	1,2-dioctanoylglycerol	61-65	formyl peptide receptor 1	Homo sapiens	128-132	
2174064-9	1990	Third, following neutrophil priming with dioctanoylglycerol (diC8), maximal O2- production occurred in response to 0.015 microM fMLP or Con A without a rise in [Ca]i, and diC8/fMLP-induced O2- production was not inhibited by EGTA.	1,2-dioctanoylglycerol	61-65	formyl peptide receptor 1	Homo sapiens	176-180