PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25663381-7	2015	Our data suggest that the activation of the hexosamine biosynthetic pathway directly increased OGT levels and activity, triggering changes in glycosylation and PASMC proliferation.	pasmc	160-165	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	95-98	
25663381-8	2015	Partial knockdown of OGT in IPAH PASMCs resulted in reduced global O-linked beta-N-acetylglucosamine modification levels and abrogated PASMC proliferation.	pasmc	33-38	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	21-24	
25663381-10	2015	CONCLUSIONS: Our data demonstrate that hexosamine biosynthetic pathway flux is increased in IPAH and drives OGT-facilitated PASMC proliferation through specific proteolysis and direct activation of host cell factor-1.	pasmc	124-129	O-linked N-acetylglucosamine (GlcNAc) transferase	Homo sapiens	108-111