PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23770347-7	2013	Furthermore, immunoblotting analysis of placental tissue harvested from our murine models revealed DMA-mediated regulation of expression of the proinflammatory cytokines IL-1beta, tumor necrosis factor alpha, and IL-6, and increased expression of the regulatory inflammatory cytokine IL-10.	dimethylacetamide	99-102	tumor necrosis factor	Mus musculus	180-207	
27782292-4	2016	Using in vitro and ex vivo models, we show that DMA suppresses secretion of pro-inflammatory cytokines in lipopolysaccharide (LPS)-induced RAW 264.7 cells, TNFalpha-challenged JEG-3 cells and LPS-stimulated human placental explants.	dimethylacetamide	48-51	tumor necrosis factor	Mus musculus	156-164	
27782292-5	2016	DMA significantly attenuated the secretion of TNFalpha, IL-6, IL-10, and granulocyte macrophage colony stimulating factor (GM-CSF) from LPS-stimulated RAW 264.7 cells, IL-6 secretion from TNFalpha-stimulated JEG-3 cells and TNFalpha, IL-6, IL-10, GM-CSF and Interleukin-8 (IL-8) from LPS-stimulated human placental explants.	dimethylacetamide	0-3	tumor necrosis factor	Mus musculus	46-54	
27782292-5	2016	DMA significantly attenuated the secretion of TNFalpha, IL-6, IL-10, and granulocyte macrophage colony stimulating factor (GM-CSF) from LPS-stimulated RAW 264.7 cells, IL-6 secretion from TNFalpha-stimulated JEG-3 cells and TNFalpha, IL-6, IL-10, GM-CSF and Interleukin-8 (IL-8) from LPS-stimulated human placental explants.	dimethylacetamide	0-3	tumor necrosis factor	Mus musculus	188-196