PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2527057-7	1989	Treatment of CMK cells with phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA) greatly enhanced the reactivity with anti-platelet antibodies, increased the number of cells in which cytoplasm was dissociated into numerous segments and suppressed the reactivity with anti-glycophorin A.	Tetradecanoylphorbol Acetate	42-78	glycophorin A (MNS blood group)	Homo sapiens	276-289	
2527057-7	1989	Treatment of CMK cells with phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA) greatly enhanced the reactivity with anti-platelet antibodies, increased the number of cells in which cytoplasm was dissociated into numerous segments and suppressed the reactivity with anti-glycophorin A.	Tetradecanoylphorbol Acetate	80-83	glycophorin A (MNS blood group)	Homo sapiens	276-289	
14769215-4	2004	When K562 cells were treated with TPA 50 microg/L for 72 h to induce megakaryocytic differentiation, the presence of chebulinic acid 50 micromol/L slightly provoked the decrease of GPA protein expression induced by TPA.	Tetradecanoylphorbol Acetate	215-218	glycophorin A (MNS blood group)	Homo sapiens	181-184	
3370227-2	1988	TPA also stimulated minor incorporation of [32 P]Pi into most integral membrane proteins, including band 3, glycophorin A, the band 4.5 region (glucose transporter) and band 7.	Tetradecanoylphorbol Acetate	0-3	glycophorin A (MNS blood group)	Homo sapiens	108-121	
3755722-9	1986	Furthermore, we show that the expression of the glycophorin A and B genes are coordinately regulated by tumor-promoting phorbol ester, 12-O-tetradecanoylphorbol-13-acetate.	Tetradecanoylphorbol Acetate	135-171	glycophorin A (MNS blood group)	Homo sapiens	48-67	
8443382-3	1993	Studies of the molecular mechanism of PMA-induced differentiation in HEL cells showed that when HEL cells are treated with PMA, they dramatically decrease the expression of the erythroid-specific gene glycophorin A at the mRNA level but apparently not at the steady-state protein level.	Tetradecanoylphorbol Acetate	38-41	glycophorin A (MNS blood group)	Homo sapiens	201-214	
7504152-2	1993	When HEL cells were treated with 10(-7) M TPA, glycophorin A expression and hemoglobin synthesis were reduced, while the expression of GP IIb/IIIa was induced in association with the morphological changes.	Tetradecanoylphorbol Acetate	42-45	glycophorin A (MNS blood group)	Homo sapiens	47-60	
8287040-2	1993	12-O-Tetradecanoylphorbol-13- acetate (TPA) suppressed the expression of the erythrocytic (glycophorin A) and myelocytic (CD11b) antigens in K562-L, but increased the expression of these antigens in K562-H. TPA increased the megakaryocytic (CD61) antigens in both cells.	Tetradecanoylphorbol Acetate	0-37	glycophorin A (MNS blood group)	Homo sapiens	91-104	
8287040-2	1993	12-O-Tetradecanoylphorbol-13- acetate (TPA) suppressed the expression of the erythrocytic (glycophorin A) and myelocytic (CD11b) antigens in K562-L, but increased the expression of these antigens in K562-H. TPA increased the megakaryocytic (CD61) antigens in both cells.	Tetradecanoylphorbol Acetate	39-42	glycophorin A (MNS blood group)	Homo sapiens	91-104