PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12646569-4	2003	Protein kinase activators (cAMP, forskolin, or phorbol-12-myristate-13-acetate) markedly increased GSTA4-4 targeting to mitochondria, whereas kinase inhibitors caused its retention in the cytosol.	Tetradecanoylphorbol Acetate	47-78	glutathione S-transferase, alpha 4	Mus musculus	99-106	
20966433-6	2010	We used quantitative polymerase chain reaction, western blotting, and immunohistochemistry to verify induction of Gsta4 in mouse epidermis following TPA treatment and biochemical assays to associate Gsta4 activity with tumor promotion susceptibility.	Tetradecanoylphorbol Acetate	149-152	glutathione S-transferase, alpha 4	Mus musculus	114-119	
20966433-10	2010	Gsta4 maps to Psl1.2 and was highly induced (mRNA and protein) in the epidermis of resistant C57BL/6 mice compared with that of sensitive DBA/2 mice following treatment with TPA.	Tetradecanoylphorbol Acetate	174-177	glutathione S-transferase, alpha 4	Mus musculus	0-5	
20966433-11	2010	Gsta4 activity levels were also higher in the epidermis of C57BL/6 mice following treatment with TPA.	Tetradecanoylphorbol Acetate	97-100	glutathione S-transferase, alpha 4	Mus musculus	0-5	
20966433-12	2010	Gsta4-deficient mice (C57BL/6.Gsta4(-/-) mice) were more sensitive to TPA skin tumor promotion (0.8 tumors per mouse vs 0.4 tumors per mouse in wild-type controls; difference = 0.4 tumors per mouse; 95% confidence interval = 0.1 to 0.7, P = .007).	Tetradecanoylphorbol Acetate	70-73	glutathione S-transferase, alpha 4	Mus musculus	0-5	
16044405-9	2005	Three genes, Gsta4, Nmes1 (MGC58382), and Serpinb2, located within promotion susceptibility loci Psl1 (chr 9), Psl2 (chr 2), and Psl3 (chr 1), respectively, were identified in this analysis as potential candidates for modifiers of susceptibility to skin tumor promotion by TPA.	Tetradecanoylphorbol Acetate	273-276	glutathione S-transferase, alpha 4	Mus musculus	13-18