PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 25543502-11 2014 The amount of vWF increased in cells, and vWF strings were formed on cell surface by the stimulation of phorbol-12-myristate-13-acetate(PMA). Tetradecanoylphorbol Acetate 104-135 von Willebrand factor Homo sapiens 14-17 25647520-2 2015 The target protein, von Willebrand factor (vWF), was released from human endothelial cells by adding phorbol-12-myristate-13-acetate (PMA). Tetradecanoylphorbol Acetate 101-132 von Willebrand factor Homo sapiens 20-41 25647520-2 2015 The target protein, von Willebrand factor (vWF), was released from human endothelial cells by adding phorbol-12-myristate-13-acetate (PMA). Tetradecanoylphorbol Acetate 101-132 von Willebrand factor Homo sapiens 43-46 25647520-2 2015 The target protein, von Willebrand factor (vWF), was released from human endothelial cells by adding phorbol-12-myristate-13-acetate (PMA). Tetradecanoylphorbol Acetate 134-137 von Willebrand factor Homo sapiens 20-41 25647520-2 2015 The target protein, von Willebrand factor (vWF), was released from human endothelial cells by adding phorbol-12-myristate-13-acetate (PMA). Tetradecanoylphorbol Acetate 134-137 von Willebrand factor Homo sapiens 43-46 25543502-11 2014 The amount of vWF increased in cells, and vWF strings were formed on cell surface by the stimulation of phorbol-12-myristate-13-acetate(PMA). Tetradecanoylphorbol Acetate 104-135 von Willebrand factor Homo sapiens 42-45 25543502-11 2014 The amount of vWF increased in cells, and vWF strings were formed on cell surface by the stimulation of phorbol-12-myristate-13-acetate(PMA). Tetradecanoylphorbol Acetate 136-139 von Willebrand factor Homo sapiens 14-17 25543502-11 2014 The amount of vWF increased in cells, and vWF strings were formed on cell surface by the stimulation of phorbol-12-myristate-13-acetate(PMA). Tetradecanoylphorbol Acetate 136-139 von Willebrand factor Homo sapiens 42-45 15061043-5 2004 RESULTS: The amount of staining with vWF (p = 0.04) and uPA (p = 0.02) showed statistically significant differences, PAI-1 (p = 0.09) and tPA (p = 0.50) showed no difference between leg ulcer and control groups. Tetradecanoylphorbol Acetate 138-141 von Willebrand factor Homo sapiens 37-40 22207689-7 2012 Regulated secretion of von Willebrand factor was induced by phorbol 12-myristate 13-acetate. Tetradecanoylphorbol Acetate 60-91 von Willebrand factor Homo sapiens 23-44 8249125-6 1993 However, at therapeutic concentrations (0.1-5 micrograms/ml) vWF release by cells stimulated with thrombin, histamine, PMA, and the calcium ionophore A23187 was enhanced by both CsA and cremophor in a concentration-dependent manner. Tetradecanoylphorbol Acetate 119-122 von Willebrand factor Homo sapiens 61-64 8950233-2 1996 We have shown previously that phorbol 12-myristate 13-acetate (PMA) induces the Dami human megakaryocytic cell line to become polyploid and to express platelet-specific proteins, including von Willebrand factor (vWF) and glycoprotein Ib (GpIb). Tetradecanoylphorbol Acetate 30-61 von Willebrand factor Homo sapiens 189-210 8950233-2 1996 We have shown previously that phorbol 12-myristate 13-acetate (PMA) induces the Dami human megakaryocytic cell line to become polyploid and to express platelet-specific proteins, including von Willebrand factor (vWF) and glycoprotein Ib (GpIb). Tetradecanoylphorbol Acetate 30-61 von Willebrand factor Homo sapiens 212-215 8950233-2 1996 We have shown previously that phorbol 12-myristate 13-acetate (PMA) induces the Dami human megakaryocytic cell line to become polyploid and to express platelet-specific proteins, including von Willebrand factor (vWF) and glycoprotein Ib (GpIb). Tetradecanoylphorbol Acetate 63-66 von Willebrand factor Homo sapiens 189-210 8950233-2 1996 We have shown previously that phorbol 12-myristate 13-acetate (PMA) induces the Dami human megakaryocytic cell line to become polyploid and to express platelet-specific proteins, including von Willebrand factor (vWF) and glycoprotein Ib (GpIb). Tetradecanoylphorbol Acetate 63-66 von Willebrand factor Homo sapiens 212-215 7693024-4 1993 When the cells were cultured with 12-o-tetradecanoylphorbol-13-acetate (TPA), the expression of TSP was enhanced and vWF was also detected, but not Fbg. Tetradecanoylphorbol Acetate 72-75 von Willebrand factor Homo sapiens 117-120 34369554-9 2021 On the other hand, AGFG2 downregulation resulted in the inhibition of vWF secretion upon Phorbol 12-myristate 13-acetate (PMA) or histamine stimulation, suggesting that AGFG2 plays a role in vWF exocytosis. Tetradecanoylphorbol Acetate 89-120 von Willebrand factor Homo sapiens 70-73 2317558-3 1990 Regulated secretion of stored vWF induced by thrombin or phorbol-12-myristate-13-acetate (PMA) was also diminished in vWD cells. Tetradecanoylphorbol Acetate 57-88 von Willebrand factor Homo sapiens 30-33 2317558-3 1990 Regulated secretion of stored vWF induced by thrombin or phorbol-12-myristate-13-acetate (PMA) was also diminished in vWD cells. Tetradecanoylphorbol Acetate 90-93 von Willebrand factor Homo sapiens 30-33 7693024-4 1993 When the cells were cultured with 12-o-tetradecanoylphorbol-13-acetate (TPA), the expression of TSP was enhanced and vWF was also detected, but not Fbg. Tetradecanoylphorbol Acetate 34-70 von Willebrand factor Homo sapiens 117-120 1530595-2 1992 Either elevation of intracellular Ca2+ concentration ([Ca2+]i) with a Ca2+ ionophore or activation of protein kinase (PK) C by phorbol 12-myristate 13-acetate caused vWf secretion, and together the agents acted synergistically. Tetradecanoylphorbol Acetate 127-158 von Willebrand factor Homo sapiens 166-169 1377415-3 1992 Stimuli used to promote secretion of prostacyclin and vWF were human alpha-thrombin, histamine, protamine sulphate, poly-L-lysine and phorbol myristate acetate. Tetradecanoylphorbol Acetate 134-159 von Willebrand factor Homo sapiens 54-57 1796389-4 1991 A positive correlation was demonstrated between vWF:Ag/FVIII:C ratio and tPA:antigen levels suggesting that both tPA and vWF:Ag could be considered as early indicators of a possible micro angiopathy occurring in preeclampsia. Tetradecanoylphorbol Acetate 73-76 von Willebrand factor Homo sapiens 48-51 34369554-9 2021 On the other hand, AGFG2 downregulation resulted in the inhibition of vWF secretion upon Phorbol 12-myristate 13-acetate (PMA) or histamine stimulation, suggesting that AGFG2 plays a role in vWF exocytosis. Tetradecanoylphorbol Acetate 89-120 von Willebrand factor Homo sapiens 191-194 34369554-9 2021 On the other hand, AGFG2 downregulation resulted in the inhibition of vWF secretion upon Phorbol 12-myristate 13-acetate (PMA) or histamine stimulation, suggesting that AGFG2 plays a role in vWF exocytosis. Tetradecanoylphorbol Acetate 122-125 von Willebrand factor Homo sapiens 70-73 34369554-9 2021 On the other hand, AGFG2 downregulation resulted in the inhibition of vWF secretion upon Phorbol 12-myristate 13-acetate (PMA) or histamine stimulation, suggesting that AGFG2 plays a role in vWF exocytosis. Tetradecanoylphorbol Acetate 122-125 von Willebrand factor Homo sapiens 191-194 2785404-6 1989 However, upon stimulation of confluent endothelial cell monolayers with phorbol myristate acetate, endothelial cells predominantly secrete vWF at the lumenal surface. Tetradecanoylphorbol Acetate 72-97 von Willebrand factor Homo sapiens 139-142 2470733-4 1989 For each agonist tested (histamine, thrombin, phorbol 12-myristate 13-acetate, and the calcium ionophore A23187) a dose-dependent redistribution of GMP-140 to the endothelial surface was observed which closely paralleled the dose-dependent secretion of vWF into the cell supernatant. Tetradecanoylphorbol Acetate 46-77 von Willebrand factor Homo sapiens 253-256 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 34-65 von Willebrand factor Homo sapiens 111-114 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 34-65 von Willebrand factor Homo sapiens 190-193 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 34-65 von Willebrand factor Homo sapiens 190-193 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 67-70 von Willebrand factor Homo sapiens 111-114 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 67-70 von Willebrand factor Homo sapiens 190-193 2784474-10 1989 Long-term incubation of HUVE with phorbol 12-myristate 13-acetate (PMA), a compound that causes release of HMW vWf from Weibel-Palade bodies in HUVE, resulted in a marked decrease in matrix vWf (0.2% to 1% of the total vWf). Tetradecanoylphorbol Acetate 67-70 von Willebrand factor Homo sapiens 190-193 3069641-12 1988 Short-term (less than 1 h) treatment of endothelial cells with the perturbing phorbol ester 4 beta-phorbol-12-myristate-13-acetate (PMA) results in release of cellular stored von Willebrand factor. Tetradecanoylphorbol Acetate 94-130 von Willebrand factor Homo sapiens 175-196 3069641-12 1988 Short-term (less than 1 h) treatment of endothelial cells with the perturbing phorbol ester 4 beta-phorbol-12-myristate-13-acetate (PMA) results in release of cellular stored von Willebrand factor. Tetradecanoylphorbol Acetate 132-135 von Willebrand factor Homo sapiens 175-196 3069641-13 1988 24-48 h after exposure to PMA the endothelial cell distribution of von Willebrand factor is changed distinctly. Tetradecanoylphorbol Acetate 26-29 von Willebrand factor Homo sapiens 67-88 3307015-6 1987 Immuno-fluorescence of PMA-treated cells suggested that vWFAg might be less granular than in control EC but the mean levels of vWF and vWFAg in the supernatant of cells incubated with PMA were not significantly different from control values. Tetradecanoylphorbol Acetate 23-26 von Willebrand factor Homo sapiens 56-59 6436246-5 1984 The phorbol ester 4 beta-phorbol 12-myristate 13-acetate (PMA) and the Ca2+ ionophore A23187 both bypass the phospholipid methylation and directly stimulate Ca2+ influx and von Willebrand factor release. Tetradecanoylphorbol Acetate 20-56 von Willebrand factor Homo sapiens 173-194 6436246-5 1984 The phorbol ester 4 beta-phorbol 12-myristate 13-acetate (PMA) and the Ca2+ ionophore A23187 both bypass the phospholipid methylation and directly stimulate Ca2+ influx and von Willebrand factor release. Tetradecanoylphorbol Acetate 58-61 von Willebrand factor Homo sapiens 173-194