PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14527959-7	2003	Pretreatment with the PKC delta-selective inhibitor rottlerin or transfection with an antisense PKC delta oligonucleotide inhibited PMA-induced cIAP-2 expression, whereas cotransfection with a PKC delta plasmid induced cIAP-2 promoter activity, which, taken together, identifies a role for PKC delta in cIAP-2 induction.	Tetradecanoylphorbol Acetate	132-135	baculoviral IAP repeat containing 3	Homo sapiens	144-150	
14527959-5	2003	We found that cIAP-2 mRNA levels were markedly increased in human colon cancer cells by treatment with the phorbol ester, phorbol-12-myristate-13-acetate (PMA), or bryostatin 1.	Tetradecanoylphorbol Acetate	122-153	baculoviral IAP repeat containing 3	Homo sapiens	14-20	
14527959-7	2003	Pretreatment with the PKC delta-selective inhibitor rottlerin or transfection with an antisense PKC delta oligonucleotide inhibited PMA-induced cIAP-2 expression, whereas cotransfection with a PKC delta plasmid induced cIAP-2 promoter activity, which, taken together, identifies a role for PKC delta in cIAP-2 induction.	Tetradecanoylphorbol Acetate	132-135	baculoviral IAP repeat containing 3	Homo sapiens	219-225	
14527959-5	2003	We found that cIAP-2 mRNA levels were markedly increased in human colon cancer cells by treatment with the phorbol ester, phorbol-12-myristate-13-acetate (PMA), or bryostatin 1.	Tetradecanoylphorbol Acetate	155-158	baculoviral IAP repeat containing 3	Homo sapiens	14-20	
14527959-6	2003	Inhibitors of the Ca2+-independent, novel PKC isoforms, but not inhibitors of MAPK, PI3-kinase, or PKA, blocked PMA-stimulated cIAP-2 mRNA expression, suggesting a role of PKC in PMA-mediated cIAP-2 induction.	Tetradecanoylphorbol Acetate	112-115	baculoviral IAP repeat containing 3	Homo sapiens	127-133	
14527959-7	2003	Pretreatment with the PKC delta-selective inhibitor rottlerin or transfection with an antisense PKC delta oligonucleotide inhibited PMA-induced cIAP-2 expression, whereas cotransfection with a PKC delta plasmid induced cIAP-2 promoter activity, which, taken together, identifies a role for PKC delta in cIAP-2 induction.	Tetradecanoylphorbol Acetate	132-135	baculoviral IAP repeat containing 3	Homo sapiens	219-225	
14527959-6	2003	Inhibitors of the Ca2+-independent, novel PKC isoforms, but not inhibitors of MAPK, PI3-kinase, or PKA, blocked PMA-stimulated cIAP-2 mRNA expression, suggesting a role of PKC in PMA-mediated cIAP-2 induction.	Tetradecanoylphorbol Acetate	112-115	baculoviral IAP repeat containing 3	Homo sapiens	192-198	
14527959-8	2003	Treatment with the proteasome inhibitor, MG132 or inhibitors of NF-kappa B (e.g. PDTC and gliotoxin), decreased PMA-induced up-regulation of cIAP-2.	Tetradecanoylphorbol Acetate	112-115	baculoviral IAP repeat containing 3	Homo sapiens	141-147	
12082637-4	2002	A RNase-protection analysis showed that PMA stimulated the expression of several known anti-apoptotic genes (TRAF1, TRAF4, c-IAP-1, c-IAP-2, Bfl-1, Bcl-xl).	Tetradecanoylphorbol Acetate	40-43	baculoviral IAP repeat containing 3	Homo sapiens	132-139