PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15313406-3	2004	In the present study we examined the inhibitory effect of several chemopreventive agents on the tumor necrosis factor (TNF) alpha- or 12-O-tetradecanoylphorbol 13 acetate (TPA)-induced promoter activity of GSTP1-1, as demonstrated by transient transfection experiments in K562 and U937 leukemia cells.	Tetradecanoylphorbol Acetate	134-170	glutathione S-transferase pi 1	Homo sapiens	206-213	
15981203-6	2005	The activities of cutaneous gamma-glutamyl transpeptidase (GGT) and glutathione-S-transferase P (GST-P), marker enzymes of tumorigenesis, were found to exhibit higher expression in AO or isolated sanguinarine/TPA treated groups when compared to control.	Tetradecanoylphorbol Acetate	209-212	glutathione S-transferase pi 1	Homo sapiens	68-95	
15981203-6	2005	The activities of cutaneous gamma-glutamyl transpeptidase (GGT) and glutathione-S-transferase P (GST-P), marker enzymes of tumorigenesis, were found to exhibit higher expression in AO or isolated sanguinarine/TPA treated groups when compared to control.	Tetradecanoylphorbol Acetate	209-212	glutathione S-transferase pi 1	Homo sapiens	97-102	
21975931-4	2011	Gstp(-/-)/Tg.AC mice exposed to the proinflammatory phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA) exhibited higher tumor incidence and multiplicity with a significant thickening of skin after treatment, illustrating hyperproliferative growth.	Tetradecanoylphorbol Acetate	66-102	glutathione S-transferase pi 1	Homo sapiens	0-4	
21975931-4	2011	Gstp(-/-)/Tg.AC mice exposed to the proinflammatory phorbol ester 12-O-tetradecanoylphorbol-13-acetate (TPA) exhibited higher tumor incidence and multiplicity with a significant thickening of skin after treatment, illustrating hyperproliferative growth.	Tetradecanoylphorbol Acetate	104-107	glutathione S-transferase pi 1	Homo sapiens	0-4	
21975931-7	2011	Within 4 weeks of TPA treatment, Gstp(-/-)/Tg.AC mice displayed altered lipid/sterol metabolism and Wnt signaling along with aberrant processes of cytoskeletal control and epidermal morphogenesis at both early and late times.	Tetradecanoylphorbol Acetate	18-21	glutathione S-transferase pi 1	Homo sapiens	33-37	
15313406-3	2004	In the present study we examined the inhibitory effect of several chemopreventive agents on the tumor necrosis factor (TNF) alpha- or 12-O-tetradecanoylphorbol 13 acetate (TPA)-induced promoter activity of GSTP1-1, as demonstrated by transient transfection experiments in K562 and U937 leukemia cells.	Tetradecanoylphorbol Acetate	172-175	glutathione S-transferase pi 1	Homo sapiens	206-213	
14623254-3	2003	In contrast, during megakaryocytic differentiation by 12-O-tetradecanoyl phorbol 13-acetate (TPA), GSTP1-1 expression decreased at both mRNA and protein levels.	Tetradecanoylphorbol Acetate	54-91	glutathione S-transferase pi 1	Homo sapiens	99-106	
14623254-3	2003	In contrast, during megakaryocytic differentiation by 12-O-tetradecanoyl phorbol 13-acetate (TPA), GSTP1-1 expression decreased at both mRNA and protein levels.	Tetradecanoylphorbol Acetate	93-96	glutathione S-transferase pi 1	Homo sapiens	99-106	
11226521-4	2001	Promoter deletion analyses revealed that the activator protein-1 (AP-1) transcription factor site was crucial for 12-O-tetradecanoyl phorbol 13-acetate (TPA)-mediated GSTP1 gene transcription.	Tetradecanoylphorbol Acetate	114-151	glutathione S-transferase pi 1	Homo sapiens	167-172	
11226521-4	2001	Promoter deletion analyses revealed that the activator protein-1 (AP-1) transcription factor site was crucial for 12-O-tetradecanoyl phorbol 13-acetate (TPA)-mediated GSTP1 gene transcription.	Tetradecanoylphorbol Acetate	153-156	glutathione S-transferase pi 1	Homo sapiens	167-172	
11226521-7	2001	These results show for the first time that the phorbol ester TPA is involved in the molecular mechanism(s) mediating the activation of the GSTP1 promoter in a human leukemia model.	Tetradecanoylphorbol Acetate	61-64	glutathione S-transferase pi 1	Homo sapiens	139-144	
1408831-1	1992	Glutathione Transferase P (GST-P) gene expression is dominantly regulated by an upstream enhancer (GPEI) consisting of a dyad of palindromically oriented imperfect TPA (12-O-tetradecanoyl-phorbol-13-acetate)-responsive elements (TRE).	Tetradecanoylphorbol Acetate	164-167	glutathione S-transferase pi 1	Homo sapiens	27-32	
1408831-1	1992	Glutathione Transferase P (GST-P) gene expression is dominantly regulated by an upstream enhancer (GPEI) consisting of a dyad of palindromically oriented imperfect TPA (12-O-tetradecanoyl-phorbol-13-acetate)-responsive elements (TRE).	Tetradecanoylphorbol Acetate	169-206	glutathione S-transferase pi 1	Homo sapiens	27-32