PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22023041-7	2012	By linear correlation analysis, GDF-15 exhibited a moderate relation to von Willebrand factor (r = 0.30), and weak, albeit significant relations (r = 0.13-0.29) to E-selectin, P-selectin, ICAM-1, VCAM-1, MMP-9, TIMP-1, D-dimer, PAI-1 activity and tPA-antigen.	Tetradecanoylphorbol Acetate	247-250	growth differentiation factor 15	Homo sapiens	32-38	
15757899-4	2005	Pretreatment with protein kinase C (PKC) inhibitor blocked the TPA-induced increase in NAG-1 protein levels and NF-kappa B binding/transcriptional activity, whereas an inhibition of p38, JNK, MEK activity had no effect on TPA-induced NAG-1 levels and NF-kappa B transcriptional activity.	Tetradecanoylphorbol Acetate	63-66	growth differentiation factor 15	Homo sapiens	234-239	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	14-17	growth differentiation factor 15	Homo sapiens	26-31	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	14-17	growth differentiation factor 15	Homo sapiens	46-51	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	14-17	growth differentiation factor 15	Homo sapiens	46-51	
15757899-2	2005	In the current study, the regulation of NAG-1 expression in LNCaP human prostate carcinoma cells by 12-O-tetradecanoylphorbol-13-acetate (TPA) was examined.	Tetradecanoylphorbol Acetate	100-136	growth differentiation factor 15	Homo sapiens	40-45	
15757899-2	2005	In the current study, the regulation of NAG-1 expression in LNCaP human prostate carcinoma cells by 12-O-tetradecanoylphorbol-13-acetate (TPA) was examined.	Tetradecanoylphorbol Acetate	138-141	growth differentiation factor 15	Homo sapiens	40-45	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	82-85	growth differentiation factor 15	Homo sapiens	26-31	
15757899-3	2005	TPA treatment increased NAG-1 protein and mRNA levels in a time- and concentration-dependent manner as well as NF-kappa B binding/transcriptional activity in LNCaP cells.	Tetradecanoylphorbol Acetate	0-3	growth differentiation factor 15	Homo sapiens	24-29	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	82-85	growth differentiation factor 15	Homo sapiens	46-51	
15757899-4	2005	Pretreatment with protein kinase C (PKC) inhibitor blocked the TPA-induced increase in NAG-1 protein levels and NF-kappa B binding/transcriptional activity, whereas an inhibition of p38, JNK, MEK activity had no effect on TPA-induced NAG-1 levels and NF-kappa B transcriptional activity.	Tetradecanoylphorbol Acetate	63-66	growth differentiation factor 15	Homo sapiens	87-92	
15757899-7	2005	Inhibition of TPA-induced NAG-1 expression by NAG-1 short interfering RNA blocked TPA-induced apoptosis in LNCaP cells, suggesting induction of NAG-1 negatively affects LNCaP cell survival.	Tetradecanoylphorbol Acetate	82-85	growth differentiation factor 15	Homo sapiens	46-51	
15757899-8	2005	These results demonstrate that NAG-1 expression is up-regulated by TPA in LNCaP cells through a PKC-dependent pathway involving the activation of NF-kappa B.	Tetradecanoylphorbol Acetate	67-70	growth differentiation factor 15	Homo sapiens	31-36	
9886240-5	1999	MIC-1 is not expressed in resting macrophages but is induced by a number of different activation agents, including phorbol myristate acetate, interleukin 1, tumor necrosis factor alpha, and macrophage colony-stimulating factor but not by lipopolysaccharide or interferon-gamma.	Tetradecanoylphorbol Acetate	115-140	growth differentiation factor 15	Homo sapiens	0-5