PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1394224-6	1992	The decrease in K-ras expression was greater for TPA-treated cells than for 1-beta-arabinofuranosylcytosine-treated cells.	Tetradecanoylphorbol Acetate	49-52	KRAS proto-oncogene, GTPase	Homo sapiens	16-21	
11304537-11	2001	In a second experiment, NS3 inhibited the oxidative burst induced by phorbol 12-myristate 13-acetate.	Tetradecanoylphorbol Acetate	69-100	KRAS proto-oncogene, GTPase	Homo sapiens	24-27	
10344756-0	1999	Activated Ki-Ras suppresses 12-O-tetradecanoylphorbol-13-acetate-induced activation of the c-Jun NH2-terminal kinase pathway in human colon cancer cells.	Tetradecanoylphorbol Acetate	28-64	KRAS proto-oncogene, GTPase	Homo sapiens	10-16	
10344756-3	1999	12-O-Tetradecanoylphorbol-13-acetate (TPA) induced immediate early genes, such as c-Jun, c-Fos, and Egr-1 in activated Ki-ras-disrupted clones, whereas c-Jun induction was rare in HCT116.	Tetradecanoylphorbol Acetate	0-36	KRAS proto-oncogene, GTPase	Homo sapiens	119-125	
10344756-3	1999	12-O-Tetradecanoylphorbol-13-acetate (TPA) induced immediate early genes, such as c-Jun, c-Fos, and Egr-1 in activated Ki-ras-disrupted clones, whereas c-Jun induction was rare in HCT116.	Tetradecanoylphorbol Acetate	38-41	KRAS proto-oncogene, GTPase	Homo sapiens	119-125	
10344756-4	1999	TPA induced both phosphorylation of stress-activated protein kinase kinase 1 (SEK1) and c-Jun NH2-terminal kinase (JNK) in the activated Ki-ras-disrupted clones but not in HCT116.	Tetradecanoylphorbol Acetate	0-3	KRAS proto-oncogene, GTPase	Homo sapiens	137-143	
10344756-5	1999	On the other hand, TPA-induced mitogen-activated protein kinase kinase 1/2 (MEK1/2)-extracellular signal-regulated kinase (ERK) activation was equally induced between HCT116 and the Ki-ras-disrupted clones.	Tetradecanoylphorbol Acetate	19-22	KRAS proto-oncogene, GTPase	Homo sapiens	182-188	
10344756-6	1999	Furthermore, TPA-induced SEK1-JNK activation was observed in a DLD-1-derived activated Ki-ras-disrupted clone but not in DLD-1.	Tetradecanoylphorbol Acetate	13-16	KRAS proto-oncogene, GTPase	Homo sapiens	87-93	
1905005-4	1991	In contrast, normal c-fos expression was induced by TPA in fibroblasts transformed by N- or K-ras or by the ras-unrelated oncogenes dbl and trk.	Tetradecanoylphorbol Acetate	52-55	KRAS proto-oncogene, GTPase	Homo sapiens	92-97	
2550470-5	1989	However, the reactivated viral K-ras protein strongly increased the stimulability of membrane-associated PKC by TPA and it further increased TPA"s ability to enhance the responsiveness of adenylate cyclase to IPR and PGE1.	Tetradecanoylphorbol Acetate	112-115	KRAS proto-oncogene, GTPase	Homo sapiens	31-36	
2550470-5	1989	However, the reactivated viral K-ras protein strongly increased the stimulability of membrane-associated PKC by TPA and it further increased TPA"s ability to enhance the responsiveness of adenylate cyclase to IPR and PGE1.	Tetradecanoylphorbol Acetate	141-144	KRAS proto-oncogene, GTPase	Homo sapiens	31-36	
2556070-4	1989	Our results represent the first induction of KRAS gene rearrangement in cells of patients with familiar polyposis coli (FPC) treated with N-methyl-N"-nitro-N-nitrosoguanidine (MNNG) and 12-0-tetradecanoyl-phorbol-13-acetate (TPA).	Tetradecanoylphorbol Acetate	225-228	KRAS proto-oncogene, GTPase	Homo sapiens	45-49