PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3654756-4	1987	There are differences in where, when, and how myofibrillar alpha-actin and MHC are degraded and eliminated from TPA-myosacs.	Tetradecanoylphorbol Acetate	112-115	major histocompatibility complex, class I, C	Homo sapiens	75-78	
1838253-9	1991	Treatment of cells known to activate the protein kinase C (TPA) and inositol triphosphate pathways has increased the level of beta-MHC mRNA while that of alpha-MHC remained unchanged.	Tetradecanoylphorbol Acetate	59-62	major histocompatibility complex, class I, C	Homo sapiens	131-134	
2469725-6	1989	Cross-linking class I MHC molecules on Jurkat cells induced a rise by [Ca2+]i and induced IL-2 production upon co-stimulation with PMA.	Tetradecanoylphorbol Acetate	131-134	major histocompatibility complex, class I, C	Homo sapiens	22-25	
3654756-16	1987	TPA reversibly blocks incorporation of [35S]methionine into myofibrillar alpha-actin, MHC, myosin light chains 1 and 2, the tropomyosins, and troponin C. It does not block the synthesis of beta- or gamma-actins, the nonmyofibrillar MHC or light chains, tubulin, vimentin, desmin, or most household molecules.	Tetradecanoylphorbol Acetate	0-3	major histocompatibility complex, class I, C	Homo sapiens	86-89	
3654756-16	1987	TPA reversibly blocks incorporation of [35S]methionine into myofibrillar alpha-actin, MHC, myosin light chains 1 and 2, the tropomyosins, and troponin C. It does not block the synthesis of beta- or gamma-actins, the nonmyofibrillar MHC or light chains, tubulin, vimentin, desmin, or most household molecules.	Tetradecanoylphorbol Acetate	0-3	major histocompatibility complex, class I, C	Homo sapiens	232-235