PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6083869-9	1984	Monocytic differentiation of the cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) and of cell line U 937 by dimethylsulfoxide and TPA was accompanied by a decrease in reactivity with these antibodies, which could be recovered by neuraminidase treatment.	Tetradecanoylphorbol Acetate	142-145	neuraminidase 1	Homo sapiens	241-254	
16835219-3	2006	In contrast to other cellular sialidases Neu2, Neu3, and Neu4, whose expression either remains unchanged or is down-regulated, Neu1 mRNA, protein and activity are specifically increased during the phorbol 12-myristate 13-acetate-induced differentiation, consistent with a significant induction of the transcriptional activity of the Neu1 gene promoter.	Tetradecanoylphorbol Acetate	197-228	neuraminidase 1	Homo sapiens	127-131	
16835219-3	2006	In contrast to other cellular sialidases Neu2, Neu3, and Neu4, whose expression either remains unchanged or is down-regulated, Neu1 mRNA, protein and activity are specifically increased during the phorbol 12-myristate 13-acetate-induced differentiation, consistent with a significant induction of the transcriptional activity of the Neu1 gene promoter.	Tetradecanoylphorbol Acetate	197-228	neuraminidase 1	Homo sapiens	333-337	
1362194-10	1992	Monocytic differentiation of the myeloid cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) was accompanied by a decrease in reactivity with the antibodies, which could be reversed by neuraminidase digestion.	Tetradecanoylphorbol Acetate	59-95	neuraminidase 1	Homo sapiens	194-207	
1362194-10	1992	Monocytic differentiation of the myeloid cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) was accompanied by a decrease in reactivity with the antibodies, which could be reversed by neuraminidase digestion.	Tetradecanoylphorbol Acetate	97-100	neuraminidase 1	Homo sapiens	194-207	
7172413-3	1982	Neuraminidase treatments caused a time- and dose-related release of sialic acid from the cells and enhanced the stimulatory effect of cholera toxin on basal and TPA-induced ODC activities as much as the monosialoganglioside GM1.	Tetradecanoylphorbol Acetate	161-164	neuraminidase 1	Homo sapiens	0-13