PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 6083869-9 1984 Monocytic differentiation of the cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) and of cell line U 937 by dimethylsulfoxide and TPA was accompanied by a decrease in reactivity with these antibodies, which could be recovered by neuraminidase treatment. Tetradecanoylphorbol Acetate 142-145 neuraminidase 1 Homo sapiens 241-254 16835219-3 2006 In contrast to other cellular sialidases Neu2, Neu3, and Neu4, whose expression either remains unchanged or is down-regulated, Neu1 mRNA, protein and activity are specifically increased during the phorbol 12-myristate 13-acetate-induced differentiation, consistent with a significant induction of the transcriptional activity of the Neu1 gene promoter. Tetradecanoylphorbol Acetate 197-228 neuraminidase 1 Homo sapiens 127-131 16835219-3 2006 In contrast to other cellular sialidases Neu2, Neu3, and Neu4, whose expression either remains unchanged or is down-regulated, Neu1 mRNA, protein and activity are specifically increased during the phorbol 12-myristate 13-acetate-induced differentiation, consistent with a significant induction of the transcriptional activity of the Neu1 gene promoter. Tetradecanoylphorbol Acetate 197-228 neuraminidase 1 Homo sapiens 333-337 1362194-10 1992 Monocytic differentiation of the myeloid cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) was accompanied by a decrease in reactivity with the antibodies, which could be reversed by neuraminidase digestion. Tetradecanoylphorbol Acetate 59-95 neuraminidase 1 Homo sapiens 194-207 1362194-10 1992 Monocytic differentiation of the myeloid cell line HL60 by 12-O-tetradecanoylphorbol-13-acetate (TPA) was accompanied by a decrease in reactivity with the antibodies, which could be reversed by neuraminidase digestion. Tetradecanoylphorbol Acetate 97-100 neuraminidase 1 Homo sapiens 194-207 7172413-3 1982 Neuraminidase treatments caused a time- and dose-related release of sialic acid from the cells and enhanced the stimulatory effect of cholera toxin on basal and TPA-induced ODC activities as much as the monosialoganglioside GM1. Tetradecanoylphorbol Acetate 161-164 neuraminidase 1 Homo sapiens 0-13