PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27564008-5	2017	An increase in PM2.5 of 10 ug/m3 was associated with a 2.5% (95% CI: 1.6-3.4%) increased risk of COPD-related ED and HA, an increase of 10 ug/m3 in NO2 was associated with a 4.2% (2.5-6.0%) increase, and an increase of 10 ug/m3 in SO2 was associated with a 2.1% (0.7-3.5%) increase.	Sulfur Dioxide	231-234	COPD	Homo sapiens	97-101	
29679365-8	2018	The findings were that the impact of SO2 content was significantly associated with the relative risk (RR) of COPD exacerbation when the exposure took place at least 30 days or longer before hospital admission (RR 1.04-1.05; p < 0.05).	Sulfur Dioxide	37-40	COPD	Homo sapiens	109-113	
29679365-12	2018	In conclusion, the study demonstrates a salient influence of a co-existing cardiovascular malady on the appearance of COPD-related respiratory exacerbations when the pollutant SO2 and NOx contents rose.	Sulfur Dioxide	176-179	COPD	Homo sapiens	118-122	
27564008-9	2017	Ambient outdoor concentrations of PM2.5, NO2, and SO2 were significantly and positively associated with both COPD-related morbidity and mortality.	Sulfur Dioxide	50-53	COPD	Homo sapiens	109-113	
27232203-12	2016	Influenza virus, cold temperatures, and increased atmospheric NO2, CO, PM10, and SO2 (but decreased O3) concentrations were identified as potential contributors to the burden of COPD exacerbations in the community.	Sulfur Dioxide	81-84	COPD	Homo sapiens	178-182	
10843983-1	2000	Air pollution as a trigger for exacerbations of COPD has been recognized for > 50 years, and has led to the development of air quality standards in many countries that substantially decreased the levels of air pollutants derived from the burning of fossil fuels, such as black smoke and sulfur dioxide.	Sulfur Dioxide	290-304	COPD	Homo sapiens	48-52	
27195597-3	2016	We found that an increase of 10 mug/m(3) in particulate matter with an aerodynamic diameter of 10 mum or less (PM10), sulfur dioxide (SO2) and nitrogen dioxide (NO2) was associated with a 1.58% (95% confidence interval (CI): 0.12-3.06%), 3.45% (95% CI: 1.30-5.66%) and 2.35% (95% CI: 0.42-4.32%) increase of COPD mortality over a lag of 0-15 days, respectively.	Sulfur Dioxide	118-132	COPD	Homo sapiens	308-312	
27195597-3	2016	We found that an increase of 10 mug/m(3) in particulate matter with an aerodynamic diameter of 10 mum or less (PM10), sulfur dioxide (SO2) and nitrogen dioxide (NO2) was associated with a 1.58% (95% confidence interval (CI): 0.12-3.06%), 3.45% (95% CI: 1.30-5.66%) and 2.35% (95% CI: 0.42-4.32%) increase of COPD mortality over a lag of 0-15 days, respectively.	Sulfur Dioxide	134-137	COPD	Homo sapiens	308-312	
27756407-7	2016	RESULTS: We found that short-term exposures to SO2 were associated with an increase in COPD exacerbation risk (odds ratio (OR) = 2.45, 95 % CI: 1.75-3.45 per 1 ppb increase) after adjustment for PM2.5.	Sulfur Dioxide	47-50	COPD	Homo sapiens	87-91	
27756407-10	2016	CONCLUSIONS: Despite living in an area with air pollution concentrations below current USEPA NAAQS, these COPD patients appeared to suffer increased risk of COPD exacerbation following short-term exposures to increased concentrations of SO2 and NO2.	Sulfur Dioxide	237-240	COPD	Homo sapiens	106-110	
27756407-10	2016	CONCLUSIONS: Despite living in an area with air pollution concentrations below current USEPA NAAQS, these COPD patients appeared to suffer increased risk of COPD exacerbation following short-term exposures to increased concentrations of SO2 and NO2.	Sulfur Dioxide	237-240	COPD	Homo sapiens	157-161	
25451635-3	2014	Pollutant exposure can also lead to AE COPD, such as NO2, SO2, ozone or particulates (PM10 and PM2.5).	Sulfur Dioxide	58-61	COPD	Homo sapiens	39-43