PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9037860-6	1996	GST-P positive foci was significantly increased in DEN + Fen groups treated with 1800 ppm or more, DEN + Feb groups treated with 1000 ppm Feb or more and DEN + Oxf groups treated with 250 ppm Oxf or more.	Fenbendazole	57-60	glutathione S-transferase pi 1	Rattus norvegicus	0-5	
20423715-1	2010	To clarify the involvement of signaling of transforming growth factor (TGF)-beta during the hepatocarcinogenesis, the immunohistochemical distribution of related molecules was analyzed in relation with liver cell lesions expressing glutathione S-transferase placental form (GST-P) during liver tumor promotion by fenbendazole, phenobarbital, piperonyl butoxide, or thioacetamide, using rats.	Fenbendazole	313-325	glutathione S-transferase pi 1	Rattus norvegicus	232-279	
19309364-2	2009	The cellular localization of related molecules was examined in liver cell foci expressing glutathione S-transferase placental form (GST-P) at the early stage of tumor promotion by fenbendazole (FB), piperonyl butoxide, or thioacetamide.	Fenbendazole	180-192	glutathione S-transferase pi 1	Rattus norvegicus	90-137	
27914986-4	2017	Among them, TMEM70 and UBE2E2 showed increased incidences of negative foci in GST-P+ foci by promotion of all examined TAA, beta-naphthoflavone, piperonyl butoxide, fenbendazole and phenobarbital, while HIST1H2AA and SLK did not respond to all promotive treatments.	Fenbendazole	165-177	glutathione S-transferase pi 1	Rattus norvegicus	78-83