PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30089726-13	2018	CONCLUSIONS: Our results suggest that CFTR-independent mechanisms increase ASL pH in people with CF.	acetylsalicylic acid lysinate	75-78	CF transmembrane conductance regulator	Homo sapiens	38-42	
19364771-4	2009	In well differentiated human airway epithelial cells, ASL [K(+)] was 20.8 +/- 0.3 mm and decreased by inhibition of the Na(+)/K(+) pump (ouabain), ENaC (amiloride), CF transmembrane conductance regulator (CFTR(inh)-172), or K(+) channels (TEA or XE991).	acetylsalicylic acid lysinate	54-57	CF transmembrane conductance regulator	Homo sapiens	205-209	
29259090-9	2017	Together, these findings suggest that SLC6A14 activity plays a role in the modification of the initial stages of airway infection by altering the level of l-arginine in the ASL, which in turn affects the attachment of P. aeruginosaIMPORTANCE CF patients with shared CFTR gene mutations show significant variability in their clinical presentation of infectious lung disease.	acetylsalicylic acid lysinate	173-176	CF transmembrane conductance regulator	Homo sapiens	266-270	
26528246-8	2015	Cystic fibrosis transmembrane conductance regulator (CFTR) plays a pivotal role in regulating the periciliary ASL.	acetylsalicylic acid lysinate	110-113	CF transmembrane conductance regulator	Homo sapiens	0-51	
26528246-8	2015	Cystic fibrosis transmembrane conductance regulator (CFTR) plays a pivotal role in regulating the periciliary ASL.	acetylsalicylic acid lysinate	110-113	CF transmembrane conductance regulator	Homo sapiens	53-57