PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19210337-1	2009	BACKGROUND AND OBJECTIVE: Prostaglandin E(2), which exerts its actions via EP receptors (EP1, EP2, EP3 and EP4), is a bioactive metabolite of arachidonic acid produced by cyclooxygenase-1 and/or cyclooxygenase-2.	Arachidonic Acid	142-158	prostaglandin E receptor 3	Homo sapiens	99-102	
11201045-1	2000	Prostaglandin E2 (PGE2), which exerts its actions via EP receptors (EP1, EP2, EP3, and EP4), is a bioactive metabolite of arachidonic acid produced by cyclooxygenase (COX)-1 and/or COX-2.	Arachidonic Acid	122-138	prostaglandin E receptor 3	Homo sapiens	78-81	
25865705-0	2015	Exogenous arachidonic acid mediates permeability of human brain microvessel endothelial cells through prostaglandin E2 activation of EP3 and EP4 receptors.	Arachidonic Acid	10-26	prostaglandin E receptor 3	Homo sapiens	133-136	
25865705-8	2015	EP3 and EP4 receptor antagonists attenuated the ARA-mediated permeability of HBMEC.	Arachidonic Acid	48-51	prostaglandin E receptor 3	Homo sapiens	0-3	
25865705-9	2015	The results indicate that ARA increases permeability of HBMEC monolayers likely via increased production of PGE2 which acts upon EP3 and EP4 receptors to mediate permeability.	Arachidonic Acid	26-29	prostaglandin E receptor 3	Homo sapiens	129-132	
25865705-14	2015	The mechanism was mediated, in part, through increased prostaglandin E2 production from ARA which acted upon EP3 and EP4 receptors to increase HBMEC monolayer permeability.	Arachidonic Acid	88-91	prostaglandin E receptor 3	Homo sapiens	109-112