PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6523524-5	1984	Glutathione-S-transferase (GST) activity using acrylamide and 1-chloro 2,4-dinitrobenzene (CDNB) as substrates followed the order: liver greater than kidney greater than brain greater than erythrocytes.	Acrylamide	47-57	hematopoietic prostaglandin D synthase	Rattus norvegicus	0-25	
6523524-5	1984	Glutathione-S-transferase (GST) activity using acrylamide and 1-chloro 2,4-dinitrobenzene (CDNB) as substrates followed the order: liver greater than kidney greater than brain greater than erythrocytes.	Acrylamide	47-57	hematopoietic prostaglandin D synthase	Rattus norvegicus	27-30	
6883577-3	1983	In liver both acrylamide and styrene caused an increase in lipid peroxidation and decrease in glutathione contents and activity of glutathione-S-transferase in a dose dependent manner, while in brain only acrylamide produced a decrease in glutathione content.	Acrylamide	14-24	hematopoietic prostaglandin D synthase	Rattus norvegicus	131-156	
6704549-0	1984	Effect of acrylamide on glutathione-S-transferase activity in different regions of rat brain.	Acrylamide	10-20	hematopoietic prostaglandin D synthase	Rattus norvegicus	24-49	
6883577-3	1983	In liver both acrylamide and styrene caused an increase in lipid peroxidation and decrease in glutathione contents and activity of glutathione-S-transferase in a dose dependent manner, while in brain only acrylamide produced a decrease in glutathione content.	Acrylamide	205-215	hematopoietic prostaglandin D synthase	Rattus norvegicus	131-156	
6883577-9	1983	The inhibition of glutathione-S-transferase activity by acrylamide and styrene suggests that detoxication of these neurotoxic compounds could be suppressed following acute exposure.	Acrylamide	56-66	hematopoietic prostaglandin D synthase	Rattus norvegicus	18-43	
31719782-0	2018	Glutathione S-transferase is a good biomarker in acrylamide induced neurotoxicity and genotoxicity.	Acrylamide	49-59	hematopoietic prostaglandin D synthase	Rattus norvegicus	0-25	
7222094-0	1981	Acrylamide induced inhibition of hepatic glutathione-S-transferase activity in rats.	Acrylamide	0-10	hematopoietic prostaglandin D synthase	Rattus norvegicus	41-66	
7222094-2	1981	with acrylamide (50 mg/kg/day) for 5 days showed a significant inhibition of hepatic glutathione-S-transferase (GST) activity; maximum inhibition occurred in 15-day-old rats in which early development of hind limb paralysis was noted.	Acrylamide	5-15	hematopoietic prostaglandin D synthase	Rattus norvegicus	85-110	
7222094-2	1981	with acrylamide (50 mg/kg/day) for 5 days showed a significant inhibition of hepatic glutathione-S-transferase (GST) activity; maximum inhibition occurred in 15-day-old rats in which early development of hind limb paralysis was noted.	Acrylamide	5-15	hematopoietic prostaglandin D synthase	Rattus norvegicus	112-115	
7222094-3	1981	Addition of acrylamide in vitro to the assay system also inhibited GST activity.	Acrylamide	12-22	hematopoietic prostaglandin D synthase	Rattus norvegicus	67-70	
25126019-6	2014	RESULTS: The administration of acrylamide resulted in significant elevation in testicular and epididymal malondialdehyde level (MDA) and significant reduction in the level of reduced glutathione (GSH) and the activities of glutathione-S-transferase (GST), glutathione peroxidase (GPX) and glutathione reductase (GR).	Acrylamide	31-41	hematopoietic prostaglandin D synthase	Rattus norvegicus	223-248	
25126019-6	2014	RESULTS: The administration of acrylamide resulted in significant elevation in testicular and epididymal malondialdehyde level (MDA) and significant reduction in the level of reduced glutathione (GSH) and the activities of glutathione-S-transferase (GST), glutathione peroxidase (GPX) and glutathione reductase (GR).	Acrylamide	31-41	hematopoietic prostaglandin D synthase	Rattus norvegicus	250-253	
3008946-0	1986	Effect of single and repeated doses of acrylamide and bis-acrylamide on glutathione-S-transferase and dopamine receptors in rat brain.	Acrylamide	39-49	hematopoietic prostaglandin D synthase	Rattus norvegicus	72-97	
16343728-8	2006	The concentration of thiobarbituric acid reactive substances, and the activities of glutathione S-transferase and superoxide dismutase in plasma, liver, testes, brain, and kidney were increased in acrylamide-treated rats.	Acrylamide	197-207	hematopoietic prostaglandin D synthase	Rattus norvegicus	84-109	
3008946-4	1986	In vitro, GST activity was also inhibited as a function of acrylamide concentration.	Acrylamide	59-69	hematopoietic prostaglandin D synthase	Rattus norvegicus	10-13	
3008946-6	1986	Repeated administration of either acrylamide or bis-acrylamide in rats (50 mg/kg X 10 days) decreased GSH content in the brain but GST activity was inhibited only by acrylamide and not by bis-acrylamide.	Acrylamide	34-44	hematopoietic prostaglandin D synthase	Rattus norvegicus	131-134	
3008946-6	1986	Repeated administration of either acrylamide or bis-acrylamide in rats (50 mg/kg X 10 days) decreased GSH content in the brain but GST activity was inhibited only by acrylamide and not by bis-acrylamide.	Acrylamide	52-62	hematopoietic prostaglandin D synthase	Rattus norvegicus	131-134