PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25240148-6	2014	Terbutaline suppressed T cell proliferation and IL-6 production and increased AOE activities involving ERK, PKA, PKC, and NF-kappaB pathways and NO production.	Terbutaline	0-11	mitogen-activated protein kinase 1	Homo sapiens	103-106	
28162907-0	2017	beta2-receptor agonists salbutamol and terbutaline attenuated cytokine production by suppressing ERK pathway through cAMP in macrophages.	Terbutaline	39-50	mitogen-activated protein kinase 1	Homo sapiens	97-100	
28162907-7	2017	In conclusion, we suggest that beta2-receptor agonists salbutamol and terbutaline inhibit inflammatory gene expression partly by a mechanism dependent on cAMP leading to the inhibition of ERK signaling in macrophages.	Terbutaline	70-81	mitogen-activated protein kinase 1	Homo sapiens	188-191