PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 25986610-3 2015 We show that SAF-A is phosphorylated on serine 59 in mitosis, that phosphorylation requires polo-like kinase 1 (PLK1) rather than DNA-PKcs, that SAF-A interacts with PLK1 in nocodazole-treated cells, and that serine 59 is dephosphorylated by protein phosphatase 2A (PP2A) in mitosis. Nocodazole 174-184 protein phosphatase 2 phosphatase activator Homo sapiens 266-270 16475161-5 2006 PP2A overexpression hindered or at least dramatically reduced thrombin- or nocodazole-induced F-actin stress fiber formation and microtubule (MT) dissolution. Nocodazole 75-85 protein phosphatase 2 phosphatase activator Homo sapiens 0-4 16475161-8 2006 The overexpression of PP2A significantly attenuated thrombin- or nocodazole-induced phosphorylation of HSP27 and tau, two cytoskeletal proteins, which potentially could be involved in agonist-induced cytoskeletal rearrangement and in the increase of permeability. Nocodazole 65-75 protein phosphatase 2 phosphatase activator Homo sapiens 22-26 15156565-3 2004 Okadaic acid (OA, 2-5 nM), a powerful inhibitor of protein phosphatase 2A (PP2A), significantly potentiates the effect of submaximal concentrations of nocodazole (50-200 nM) on transendothelial electrical resistance (TER) suggesting the involvement of PP2A activity in the MT-mediated EC barrier regulation. Nocodazole 151-161 protein phosphatase 2 phosphatase activator Homo sapiens 59-73 15156565-3 2004 Okadaic acid (OA, 2-5 nM), a powerful inhibitor of protein phosphatase 2A (PP2A), significantly potentiates the effect of submaximal concentrations of nocodazole (50-200 nM) on transendothelial electrical resistance (TER) suggesting the involvement of PP2A activity in the MT-mediated EC barrier regulation. Nocodazole 151-161 protein phosphatase 2 phosphatase activator Homo sapiens 75-79 15156565-3 2004 Okadaic acid (OA, 2-5 nM), a powerful inhibitor of protein phosphatase 2A (PP2A), significantly potentiates the effect of submaximal concentrations of nocodazole (50-200 nM) on transendothelial electrical resistance (TER) suggesting the involvement of PP2A activity in the MT-mediated EC barrier regulation. Nocodazole 151-161 protein phosphatase 2 phosphatase activator Homo sapiens 252-256 28562161-4 2017 In this study, we found that PP2A in association with its B56gamma regulatory subunit, are needed for the stability of BubR1 during nocodazole induced cell cycle arrest. Nocodazole 132-142 protein phosphatase 2 phosphatase activator Homo sapiens 29-33