PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9445009-4	1998	When fibroblasts from p53 knockout mice are treated with the spindle inhibitor nocodazole, a rereplication of DNA occurs without transit through mitosis.	Nocodazole	79-89	transformation related protein 53, pseudogene	Mus musculus	22-25	
18971638-5	2008	The effects of nocodazole, a well established tubulin poison, and JJ78:12 on p53 levels are remarkably similar, supporting that tubulin depolymerization is the main mechanism by which JJ78:12 treatment leads to p53 activation in cells.	Nocodazole	15-25	transformation related protein 53, pseudogene	Mus musculus	77-80	
21846807-3	2011	Our results clearly indicate that nocodazole induces senescence only in NIH3T3 cells with a functional p53 but not in I-MEF lacking a functional p53.	Nocodazole	34-44	transformation related protein 53, pseudogene	Mus musculus	103-106	
9840938-2	1998	When exposed to a spindle-disrupting drug such as nocodazole, fibroblasts derived from mice having wild-type p53 are blocked with a 4N content of DNA.	Nocodazole	50-60	transformation related protein 53, pseudogene	Mus musculus	109-112	
9448003-4	1998	However, p53 does play a critical role in nocodazole-treated cells which have exited mitotic arrest without undergoing cytokinesis and have thereby adapted.	Nocodazole	42-52	transformation related protein 53, pseudogene	Mus musculus	9-12	
9448003-5	1998	We have determined that in nocodazole-treated, adapted cells, p53 is required during a specific time window to prevent cells from reentering the cell cycle and initiating another round of DNA synthesis.	Nocodazole	27-37	transformation related protein 53, pseudogene	Mus musculus	62-65	
9448003-7	1998	The mechanism of the p53-dependent arrest in nocodazole-treated adapted cells requires the cyclin-dependent kinase inhibitor p21, as p21-/- fibroblasts fail to arrest in response to nocodazole treatment and become polyploid.	Nocodazole	45-55	transformation related protein 53, pseudogene	Mus musculus	21-24	
9448003-7	1998	The mechanism of the p53-dependent arrest in nocodazole-treated adapted cells requires the cyclin-dependent kinase inhibitor p21, as p21-/- fibroblasts fail to arrest in response to nocodazole treatment and become polyploid.	Nocodazole	182-192	transformation related protein 53, pseudogene	Mus musculus	21-24	
8761295-5	1996	In addition to engaging the apoptotic machinery, the tamoxifen-activated fusion protein exhibited other functions characteristic of wild-type p53, such as induction of WAF1 and MDM2 gene expression and activation of the p53-dependent spindle checkpoint in cells treated with nocodazole.	Nocodazole	275-285	transformation related protein 53, pseudogene	Mus musculus	142-145	
32912294-9	2020	We describe a class of dormant, p53 bound enhancers that gain H3K27ac under specific conditions, such as after treatment with Nocodazol, or transiently during reprogramming from fibroblasts to pluripotency.	Nocodazole	126-135	transformation related protein 53, pseudogene	Mus musculus	32-35