PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10348819-10 1999 Nocodazole treatment of constitutive IGF-II-expressing cells stimulated p21 expression in the presence of hyperphosphorylated pRb. Nocodazole 0-10 H3 histone pseudogene 16 Homo sapiens 72-75 22022534-5 2011 METHODOLOGY: During induction of senescence by low levels of endogenous p53 and ectopic p21, cells were co-treated with nocodazole, which eliminated proliferating cells. Nocodazole 120-130 H3 histone pseudogene 16 Homo sapiens 88-91 11376010-3 2001 Cells transiently exposed to nocodazole continue to express high levels of p53 and p21 in the cell cycle that follows the transient exposure to nocodazole and become arrested in G(1), regardless of whether they carry a diploid or polyploid genome after mitotic exit. Nocodazole 29-39 H3 histone pseudogene 16 Homo sapiens 83-86 11376010-3 2001 Cells transiently exposed to nocodazole continue to express high levels of p53 and p21 in the cell cycle that follows the transient exposure to nocodazole and become arrested in G(1), regardless of whether they carry a diploid or polyploid genome after mitotic exit. Nocodazole 144-154 H3 histone pseudogene 16 Homo sapiens 83-86 30482390-6 2019 Thus, the P/CAF-MDM2-p53-p21 axis enables the escape from mitotic cell death and confers resistance to nocodazole in HCT116(p53+/+) cells with SIRT2 suppression. Nocodazole 103-113 H3 histone pseudogene 16 Homo sapiens 25-28 25405759-7 2014 The mitotic index and the expression of p21, BubR1 and Securin were all reduced following Nocodazole treatment. Nocodazole 90-100 H3 histone pseudogene 16 Homo sapiens 40-43