PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10348819-10	1999	Nocodazole treatment of constitutive IGF-II-expressing cells stimulated p21 expression in the presence of hyperphosphorylated pRb.	Nocodazole	0-10	H3 histone pseudogene 16	Homo sapiens	72-75	
22022534-5	2011	METHODOLOGY: During induction of senescence by low levels of endogenous p53 and ectopic p21, cells were co-treated with nocodazole, which eliminated proliferating cells.	Nocodazole	120-130	H3 histone pseudogene 16	Homo sapiens	88-91	
11376010-3	2001	Cells transiently exposed to nocodazole continue to express high levels of p53 and p21 in the cell cycle that follows the transient exposure to nocodazole and become arrested in G(1), regardless of whether they carry a diploid or polyploid genome after mitotic exit.	Nocodazole	29-39	H3 histone pseudogene 16	Homo sapiens	83-86	
11376010-3	2001	Cells transiently exposed to nocodazole continue to express high levels of p53 and p21 in the cell cycle that follows the transient exposure to nocodazole and become arrested in G(1), regardless of whether they carry a diploid or polyploid genome after mitotic exit.	Nocodazole	144-154	H3 histone pseudogene 16	Homo sapiens	83-86	
30482390-6	2019	Thus, the P/CAF-MDM2-p53-p21 axis enables the escape from mitotic cell death and confers resistance to nocodazole in HCT116(p53+/+) cells with SIRT2 suppression.	Nocodazole	103-113	H3 histone pseudogene 16	Homo sapiens	25-28	
25405759-7	2014	The mitotic index and the expression of p21, BubR1 and Securin were all reduced following Nocodazole treatment.	Nocodazole	90-100	H3 histone pseudogene 16	Homo sapiens	40-43