PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23620395-6	2013	In addition, we treated Ins2(+/Akita) mice with metformin, which activates AMP-activated protein kinase (AMPK) and thereby slows the degradation of GTPCH I; despite blood glucose levels that were similar to untreated mice, those treated with metformin had significantly less albuminuria.	Metformin	48-57	GTP cyclohydrolase 1	Mus musculus	148-155	
23620395-6	2013	In addition, we treated Ins2(+/Akita) mice with metformin, which activates AMP-activated protein kinase (AMPK) and thereby slows the degradation of GTPCH I; despite blood glucose levels that were similar to untreated mice, those treated with metformin had significantly less albuminuria.	Metformin	242-251	GTP cyclohydrolase 1	Mus musculus	148-155	
19528375-7	2009	Metformin treatment also normalized acetylcholine-induced endothelial relaxation and increased the levels of GTPCH I and BH4.	Metformin	0-9	GTP cyclohydrolase 1	Mus musculus	109-114