PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31894313-0	2020	Metformin alleviates endometrial hyperplasia through the UCA1/miR-144/TGF-beta1/AKT signaling pathway.	Metformin	0-9	microRNA 144	Homo sapiens	62-69	
31894313-6	2020	By contrast, Met reduced cell viability, promoted cell apoptosis, and reduced the expression levels of UCA1, TGF-beta and p-AKT, while upregulating the expression of miR-144 and active Caspase-3 in a dose-dependent manner.	Metformin	13-16	microRNA 144	Homo sapiens	166-173	
31894313-11	2020	In conclusion, the current study suggested that Met reduces the risk of EH by reducing the expression levels of UCA1, TGF-beta and p-AKT, while increasing the levels of miR-144 and active Caspase-3 in a dose-dependent manner.	Metformin	48-51	microRNA 144	Homo sapiens	169-176