PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24791887-9	2014	Both genes and protein expression of NF-kappaB, MCP-1, ICAM-1, TGF-beta1 of MCs induced by high glucose were markedly reduced after metformin treatment in a dose-dependent manner (P < 0.05).	Metformin	132-141	intercellular adhesion molecule 1	Rattus norvegicus	55-61	
31357226-7	2019	Consequently, metformin reduced E-selectin as well ICAM and VCAM-1.	Metformin	14-23	intercellular adhesion molecule 1	Rattus norvegicus	51-55	
24791887-11	2014	Conlusion Metformin can suppress the expression of NF-kappaB, MCP-1, ICAM-1 and TGF-beta1 of glomerular MCs induced by high glucose via AMPK activation, which may partly contribute to its reno-protection.	Metformin	10-19	intercellular adhesion molecule 1	Rattus norvegicus	69-75	
33504231-7	2021	Metformin also induced a significant decrease in Plasma SIP, SPHK1 activity, inflammatory, oxidative stress markers, ICAM-1 and Caspase-3 genes expression compared to oxazolone group.	Metformin	0-9	intercellular adhesion molecule 1	Rattus norvegicus	117-123