PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 24791887-9 2014 Both genes and protein expression of NF-kappaB, MCP-1, ICAM-1, TGF-beta1 of MCs induced by high glucose were markedly reduced after metformin treatment in a dose-dependent manner (P < 0.05). Metformin 132-141 intercellular adhesion molecule 1 Rattus norvegicus 55-61 31357226-7 2019 Consequently, metformin reduced E-selectin as well ICAM and VCAM-1. Metformin 14-23 intercellular adhesion molecule 1 Rattus norvegicus 51-55 24791887-11 2014 Conlusion Metformin can suppress the expression of NF-kappaB, MCP-1, ICAM-1 and TGF-beta1 of glomerular MCs induced by high glucose via AMPK activation, which may partly contribute to its reno-protection. Metformin 10-19 intercellular adhesion molecule 1 Rattus norvegicus 69-75 33504231-7 2021 Metformin also induced a significant decrease in Plasma SIP, SPHK1 activity, inflammatory, oxidative stress markers, ICAM-1 and Caspase-3 genes expression compared to oxazolone group. Metformin 0-9 intercellular adhesion molecule 1 Rattus norvegicus 117-123