PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18640112-6	2008	The antioxidant, N-acetyl-L-cysteine (NAC), and secreted soluble mediators of RPE cells were appropriate to attenuate the effects of tBH-mediated oxidative stress.	tert-Butylhydroperoxide	133-136	X-linked Kx blood group	Homo sapiens	38-41	
31485293-8	2019	In both AMD and No AMD cells, NAC pretreatment reduced t-BHP-induced ROS production and protected from H2O2-induced cell death and ATP depletion.	tert-Butylhydroperoxide	55-60	X-linked Kx blood group	Homo sapiens	30-33	
10711676-11	2000	Glutathione and NAC completely abrogated tBH-induced increase in FasL and Fas expression and apoptosis.	tert-Butylhydroperoxide	41-44	X-linked Kx blood group	Homo sapiens	16-19	
17200984-9	2008	The in vitro exposure of human red blood cells to NAC increased the cellular low-molecular-weight thiol levels and retarded tert-butylhydroperoxide-induced cellular thiol depletion and membrane lipid peroxidation as well as effectively inhibited hypochlorous acid-induced erythrocyte lysis.	tert-Butylhydroperoxide	124-147	X-linked Kx blood group	Homo sapiens	50-53