PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26576227-3	2015	Our results indicated that Lico A significantly inhibited t-BHP-induced cytotoxicity, apoptosis, and reactive oxygen species (ROS) generation and reduced glutathione (GSH) depletion but increased the glutamate-cysteine ligase modifier (GCLM) subunit and the glutamate-cysteine ligase catalytic (GCLC) subunit genes expression.	tert-Butylhydroperoxide	58-63	glutamate-cysteine ligase, catalytic subunit	Mus musculus	295-299	
11032771-5	2000	Furthermore, while diethyl maleate (DEM) exposure to deplete cellular glutathione did not induce expression of GLCL-H, exposure to tertiary-butyl hydroperoxide (tBH), an oxidizing agent, resulted in significant upregulation of GLCL-H expression in two-cell embryos.	tert-Butylhydroperoxide	131-159	glutamate-cysteine ligase, catalytic subunit	Mus musculus	227-233	
11032771-5	2000	Furthermore, while diethyl maleate (DEM) exposure to deplete cellular glutathione did not induce expression of GLCL-H, exposure to tertiary-butyl hydroperoxide (tBH), an oxidizing agent, resulted in significant upregulation of GLCL-H expression in two-cell embryos.	tert-Butylhydroperoxide	161-164	glutamate-cysteine ligase, catalytic subunit	Mus musculus	227-233