PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10601669-9	2000	In situ hybridization showed that RTX treatment caused a marked and significant increase in the number of dorsal root ganglion (DRG) neurone profiles expressing CCKB receptor mRNA, whereas only a small increase was observed for CCKA receptor mRNA expressing neurone profiles.	resiniferatoxin	34-37	cholecystokinin B receptor	Rattus norvegicus	161-174	
10601669-12	2000	Since CCK functions as a physiological antagonist of morphine, it is suggested that RTX treatment enhances the activity of spinal CCK systems, leading to the reduced effect of morphine and increased effect of the CCKB receptor antagonist CI-988.	resiniferatoxin	84-87	cholecystokinin B receptor	Rattus norvegicus	213-226