PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20709907-6	2010	We further demonstrate that increased miR-144 is associated with reduced NRF2 levels in HbSS reticulocytes and with decreased glutathione regeneration and attenuated antioxidant capacity in HbSS erythrocytes, thereby providing a possible mechanism for the reduced oxidative stress tolerance and increased anemia severity seen in HbSS patients.	Glutathione	126-137	microRNA 144	Homo sapiens	38-45	
31243572-10	2019	This study reveals that miR-144 expression was upregulated, whereas NRF2 expression and glutathione levels were decreased in comparison with the untreated condition after miR mimic transfection, while the reduction of miR-144 expression contributed to the increased NRF2 expression and glutathione level compared with the untreated condition after miR inhibitor transfection.	Glutathione	286-297	microRNA 144	Homo sapiens	218-225	
28663050-2	2017	Nuclear factor erythroid 2-related factor 2 (NRF2), a potential target of miR-144, is a central regulator of antioxidant response, and plays an important role in glutathione (GSH) biosynthesis and recycling.	Glutathione	162-173	microRNA 144	Homo sapiens	74-81	
28663050-2	2017	Nuclear factor erythroid 2-related factor 2 (NRF2), a potential target of miR-144, is a central regulator of antioxidant response, and plays an important role in glutathione (GSH) biosynthesis and recycling.	Glutathione	175-178	microRNA 144	Homo sapiens	74-81	
28663050-6	2017	GSH and glutathion peroxidase (GPX) activities were detected to reveal the effect of miR-144 on GSH accumulation.	Glutathione	96-99	microRNA 144	Homo sapiens	85-92	
28663050-8	2017	In oxidative stress conditions, miR-144 increased the intracellular accumulation of ROS, reduced cell viability, reduced the activities of GSH and antioxidant enzymes, GPX1, and decreased the expression of GCLC, GCLM, GR and NRF2.	Glutathione	139-142	microRNA 144	Homo sapiens	32-39	
28663050-9	2017	In conclusion, miR-144 modulates oxidative stress tolerance by regulating NRF2 expression and GSH generation, which may contribute to the pathogenesis of AD.	Glutathione	94-97	microRNA 144	Homo sapiens	15-22	
31243572-8	2019	In alpha-thalassemia, miR-144 and NRF2 target are associated with glutathione level and anemia severity.	Glutathione	66-77	microRNA 144	Homo sapiens	22-29