PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23889203-7	2013	Cytotoxicity (uptake of propidium iodide) was assessed in the pyramidal cell layer of the CA1 region.	Propidium	24-40	carbonic anhydrase 1	Rattus norvegicus	90-93	
26212551-4	2015	Antimycin-A (0.1muM) and lipopolysaccharide (1ng/ml) caused low neurotoxicity on their own, measured as propidium iodide fluorescence in CA1 and CA3 regions.	Propidium	104-120	carbonic anhydrase 1	Rattus norvegicus	137-140	
30322800-4	2018	Treatment with ouabain, a Na+, K+-ATPase inhibitor, increased the ratio of propidium iodide-positive cells among NeuN-positive cells in the hippocampal CA1 region, which was prevented by MK-801 and d-AP5, specific blockers of the N-methyl-d-aspartate (NMDA) receptor.	Propidium	75-91	carbonic anhydrase 1	Rattus norvegicus	152-155	
17988655-8	2007	Subsequent cell death in the CA1 region was quantified using propidium iodide staining.	Propidium	61-77	carbonic anhydrase 1	Rattus norvegicus	29-32	
21453436-4	2011	The extent of CA1 cell injury was assessed 3 h after the end of 7 min of OGD by propidium iodide staining.	Propidium	80-96	carbonic anhydrase 1	Rattus norvegicus	14-17	
18606174-3	2008	Both protocols attenuated CA1 neuronal injury, as revealed 24h later by measuring the intensity of propidium iodide fluorescence in this region.	Propidium	99-115	carbonic anhydrase 1	Rattus norvegicus	26-29	
19759279-6	2009	Cytotoxicity was assessed by fluorescent microscopy and quantification of propidium iodide (PI) uptake in the pyramidal cell layers of the CA1 and CA3 regions and the granule cell layer of the dentate gyrus (DG).	Propidium	74-90	carbonic anhydrase 1	Rattus norvegicus	139-142	
19422385-6	2009	The extent of CA1 cell injury was assessed 3 h after the end of 7 min OGD by propidium iodide.	Propidium	77-93	carbonic anhydrase 1	Rattus norvegicus	14-17	
17363173-10	2007	OGD or glutamate (3.5-5.0 mM) damaged CA1 pyramidal neurons as assessed by propidium iodide (PI) fluorescence.	Propidium	75-91	carbonic anhydrase 1	Rattus norvegicus	38-41	
17524372-4	2007	Exposure of slice cultures to NMDA for 24-h produced increases in uptake of the non-vital fluorescent marker propidium iodide (PI) of 150-500% above control levels, most notably, in the CA1 region pyramidal cell layer.	Propidium	109-125	carbonic anhydrase 1	Rattus norvegicus	186-189	
15731600-3	2005	METHODS: Death of CA1, CA3, and dentate neurons in rat hippocampal slice cultures was assessed by propidium iodide fluorescence 48-72 h after 60-75 min of hypoxia.	Propidium	98-114	carbonic anhydrase 1	Rattus norvegicus	18-21	
15733770-8	2005	CA1 regional cell death was quantified by propidium iodide (PI) fluorescence.	Propidium	42-58	carbonic anhydrase 1	Rattus norvegicus	0-3	
16344151-10	2005	Judged by propidium iodide uptake, a selective CA1 lesion, with only minor affection on CA3, occurred in cultures exposed to oxygen-glucose deprivation for 30 min.	Propidium	10-26	carbonic anhydrase 1	Rattus norvegicus	47-50	
12742082-7	2003	The increased cell death, assessed by propidium iodide (PI) uptake, occurred not only among the most susceptible CA1 pyramidal cells, but also in CA3 and fascia dentata.	Propidium	38-54	carbonic anhydrase 1	Rattus norvegicus	113-116	
21189587-3	2003	Propidium iodide (PI) assay was used to observe the neuronal injury in CA1 and dentate gyrus (DG).	Propidium	0-16	carbonic anhydrase 1	Rattus norvegicus	71-74