PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10221756-1	1999	In line with the idea that cholecystokinin (CCK) is involved in anxiety-related behaviours, previous investigations showed that stressful conditions and an "anxiogenic" drug, yohimbine, increased the cortical release of CCK like-material (CCKLM) in awake rats, and that this effect could be prevented by diazepam.	Diazepam	304-312	cholecystokinin	Rattus norvegicus	27-42	
10221756-1	1999	In line with the idea that cholecystokinin (CCK) is involved in anxiety-related behaviours, previous investigations showed that stressful conditions and an "anxiogenic" drug, yohimbine, increased the cortical release of CCK like-material (CCKLM) in awake rats, and that this effect could be prevented by diazepam.	Diazepam	304-312	cholecystokinin	Rattus norvegicus	44-47	
10221756-1	1999	In line with the idea that cholecystokinin (CCK) is involved in anxiety-related behaviours, previous investigations showed that stressful conditions and an "anxiogenic" drug, yohimbine, increased the cortical release of CCK like-material (CCKLM) in awake rats, and that this effect could be prevented by diazepam.	Diazepam	304-312	cholecystokinin	Rattus norvegicus	220-223	
9712181-0	1998	Regulation of the action of the novel cholecystokinin-releasing peptide diazepam binding inhibitor by inhibitory hormones and taurocholate.	Diazepam	72-80	cholecystokinin	Rattus norvegicus	38-53	
10070046-0	1999	Diazepam-binding inhibitor33-50 elicits Ca2+ oscillation and CCK secretion in STC-1 cells via L-type Ca2+ channels.	Diazepam	0-8	cholecystokinin	Rattus norvegicus	61-64	
8755579-8	1996	Similarly, diazepam binding inhibitor33-52 [corrected] also stimulated CCK release and pancreatic secretion in a dose-dependent manner although it was 100 times less potent than the whole peptide.	Diazepam	11-19	cholecystokinin	Rattus norvegicus	71-74	
8780034-0	1996	Stress- and yohimbine-induced release of cholecystokinin in the frontal cortex of the freely moving rat: prevention by diazepam but not ondansetron.	Diazepam	119-127	cholecystokinin	Rattus norvegicus	41-56	
8491257-2	1993	In cerebral cortex and a subpopulation of hippocampal neurones, CCK mRNA levels were increased after a single injection of diazepam and 24 h after withdrawal from chronic diazepam treatment, but not after chronic diazepam treatment.	Diazepam	123-131	cholecystokinin	Rattus norvegicus	64-67	
8491257-2	1993	In cerebral cortex and a subpopulation of hippocampal neurones, CCK mRNA levels were increased after a single injection of diazepam and 24 h after withdrawal from chronic diazepam treatment, but not after chronic diazepam treatment.	Diazepam	171-179	cholecystokinin	Rattus norvegicus	64-67	
8491257-2	1993	In cerebral cortex and a subpopulation of hippocampal neurones, CCK mRNA levels were increased after a single injection of diazepam and 24 h after withdrawal from chronic diazepam treatment, but not after chronic diazepam treatment.	Diazepam	171-179	cholecystokinin	Rattus norvegicus	64-67	
2873044-6	1986	Higher concentration of the same drugs and diazepam (1 and 10 microM) which has high affinity for benzodiazepine receptors on gastrointestinal muscle, inhibited responses of ileum and gall-bladder to both CCK and acetylcholine.	Diazepam	43-51	cholecystokinin	Rattus norvegicus	205-208	
7699563-0	1994	Gastrointestinal effects of diazepam-withdrawal are linked to activation of central cholecystokinin-ergic pathways in rats.	Diazepam	28-36	cholecystokinin	Rattus norvegicus	84-99	
7699563-5	1994	It is concluded that in rats precipitated diazepam-withdrawal altered intestinal motility and colonic transit and that these effects are mediated by central release of cholecystokinin (CCK) or activation of CCK-ergic neurons.	Diazepam	42-50	cholecystokinin	Rattus norvegicus	168-183	
7699563-5	1994	It is concluded that in rats precipitated diazepam-withdrawal altered intestinal motility and colonic transit and that these effects are mediated by central release of cholecystokinin (CCK) or activation of CCK-ergic neurons.	Diazepam	42-50	cholecystokinin	Rattus norvegicus	185-188	
7699563-5	1994	It is concluded that in rats precipitated diazepam-withdrawal altered intestinal motility and colonic transit and that these effects are mediated by central release of cholecystokinin (CCK) or activation of CCK-ergic neurons.	Diazepam	42-50	cholecystokinin	Rattus norvegicus	207-210	
1975695-4	1990	Both CCK-B antagonists were able to suppress the withdrawal anxiogenesis and produce an anxiolytic effect in mice previously made tolerant to diazepam.	Diazepam	142-150	cholecystokinin	Rattus norvegicus	5-8