PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31735555-12	2019	INTERPRETATION: We determined that H19 might serve as a facilitator in the process of CaOx nephrocalcinosis-induced oxidative stress and renal tubular epithelial cell injury, and we revealed that the interaction between H19 and miR-216b could exert its effect via the HMGB1/TLR4/NF-kappaB pathway.	Calcium Oxalate	86-90	H19 imprinted maternally expressed transcript	Homo sapiens	35-38	
31735555-0	2019	H19 promote calcium oxalate nephrocalcinosis-induced renal tubular epithelial cell injury via a ceRNA pathway.	Calcium Oxalate	12-27	H19 imprinted maternally expressed transcript	Homo sapiens	0-3	
31735555-2	2019	A recent genome-wide gene expression profile analysis of Randall"s plaques in CaOx stone patients revealed that the expression of the long noncoding RNA H19 was significantly upregulated.	Calcium Oxalate	78-82	H19 imprinted maternally expressed transcript	Homo sapiens	153-156	
31735555-10	2019	Moreover, H19 downregulation inhibited HMGB1, TLR4 and NF-kappaB expression and suppressed CaOx nephrocalcinosis-induced renal tubular epithelial cell injury, NADPH oxidase, and oxidative stress in vivo and in vitro.	Calcium Oxalate	91-95	H19 imprinted maternally expressed transcript	Homo sapiens	10-13