PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30594845-4	2019	Our results revealed significant decrease in the mutant heterozygous genotype AG and also in IL-17F mutant allele G frequency in ITP patient group and associated with increased risk for ITP compared with the control group (P = 0.04 and P = 0.005 respectively).	Inosine Triphosphate	129-132	interleukin 17F	Homo sapiens	93-99	
21615796-5	2011	RESULTS: Compared with the control group, patients with chronic ITP had a significantly lower frequency of the IL-17F 7488CC genotype (0% vs. 4.8%, P<0.05).	Inosine Triphosphate	64-67	interleukin 17F	Homo sapiens	111-117	
21615796-6	2011	The number of IL-17F 7488C alleles among the patients with chronic ITP was also significantly lower than in the control group (8.7% vs. 15.2% OR=0.48, 95%CI=0.27-0.84, P=0.016).	Inosine Triphosphate	67-70	interleukin 17F	Homo sapiens	14-20	
21615796-8	2011	CONCLUSION: These findings suggest that the IL-17F 7488 T allele is significantly associated with the development of chronic ITP, suggesting a role for IL-17F in the pathogenesis of chronic ITP.	Inosine Triphosphate	125-128	interleukin 17F	Homo sapiens	44-50	
21615796-8	2011	CONCLUSION: These findings suggest that the IL-17F 7488 T allele is significantly associated with the development of chronic ITP, suggesting a role for IL-17F in the pathogenesis of chronic ITP.	Inosine Triphosphate	125-128	interleukin 17F	Homo sapiens	152-158