PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23140507-1	2013	OBJECTIVE: This study examines the effect of Dextromethorphan (d-3-methoxy-17-methylmorphinan; DXM), a commonly used cough-suppressing drug, on the expression of VCAM-1 and ICAM-1 in human umbilical vein endothelial cells (HUVECs) stimulated with lipopolysaccharide (LPS).	Dextromethorphan	45-61	vascular cell adhesion molecule 1	Homo sapiens	162-168	
23140507-1	2013	OBJECTIVE: This study examines the effect of Dextromethorphan (d-3-methoxy-17-methylmorphinan; DXM), a commonly used cough-suppressing drug, on the expression of VCAM-1 and ICAM-1 in human umbilical vein endothelial cells (HUVECs) stimulated with lipopolysaccharide (LPS).	Dextromethorphan	95-98	vascular cell adhesion molecule 1	Homo sapiens	162-168	
23140507-5	2013	Furthermore, treatment of HUVECs with DXM can significantly decrease LPS-induced expression of ICAM-1 and VCAM-1.	Dextromethorphan	38-41	vascular cell adhesion molecule 1	Homo sapiens	106-112	
23140507-9	2013	CONCLUSIONS: Dextromethorphan inhibits the adhesive capacity of HUVECs by reducing the LPS-induced ICAM-1 and VCAM-1 expression via the suppression of the ERK, Akt, and NF-kappaB signaling pathways.	Dextromethorphan	13-29	vascular cell adhesion molecule 1	Homo sapiens	110-116