PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35154579-8	2022	ABT199/venetocalx upregulated anti-apoptotic MCL1 as a compensatory mechanism but addition of (Bu+4HC) or (Bu+Flu) negated this effect by CASPASE 3-mediated cleavage of MEK1/2 and its substrate MCL1.	Busulfan	107-109	caspase 3	Homo sapiens	138-147	
17264767-8	2007	In addition, treatment of cells with busulfan resulted in a further increased apoptotic rate, as determined by fluorescence-activated cell sorter analysis and caspase-3 activation.	Busulfan	37-45	caspase 3	Homo sapiens	159-168