PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19740703-9	2009	PCFT mutations should be considered in infants with SCID-like phenotype, as the immunodeficiency is reversible with parenteral folinic acid repletion.	Leucovorin	127-139	solute carrier family 46 member 1	Homo sapiens	0-4	
26193446-8	2015	We conclude that SLC46A1/PCFT and SLC19A1/RFC-1 are associated with DFS of patients with colorectal cancer and hypothesize that poor response to 5-fluorouracil plus leucovorin therapy in some patients may be linked to low expression of these genes.	Leucovorin	165-175	solute carrier family 46 member 1	Homo sapiens	17-24	
26193446-8	2015	We conclude that SLC46A1/PCFT and SLC19A1/RFC-1 are associated with DFS of patients with colorectal cancer and hypothesize that poor response to 5-fluorouracil plus leucovorin therapy in some patients may be linked to low expression of these genes.	Leucovorin	165-175	solute carrier family 46 member 1	Homo sapiens	25-29	
26006721-6	2015	Although difficult, early detection and diagnosis of cerebral folate deficiency are important because folinic acid at a pharmacologic dose may normalize outcome in PCFT gene defects, as well as bypass autoantibody-blocked folate receptors and enter the cerebrospinal fluid by way of the reduced folate carrier.	Leucovorin	102-114	solute carrier family 46 member 1	Homo sapiens	164-168	
19403800-7	2009	Consistent with the intrinsic functionality of E185A-PCFT, [(3)H]MTX influx at pH 5.5 or 7.4 was trans-stimulated in cells preloaded with nonlabeled MTX or 5-formyltetrahydrofolate.	Leucovorin	156-180	solute carrier family 46 member 1	Homo sapiens	53-57	
18718264-1	2008	We report 2 sequential homozygous mutations in the recently cloned proton-coupled folate transporter (PCFT) gene, resulting in the absence of this protein, in a 27-year-old woman with hereditary folate malabsorption, normal in all respects having completed higher education, who has been treated with parenteral 5-formyltetrahydrofolate since infancy.	Leucovorin	312-336	solute carrier family 46 member 1	Homo sapiens	67-100	
18718264-1	2008	We report 2 sequential homozygous mutations in the recently cloned proton-coupled folate transporter (PCFT) gene, resulting in the absence of this protein, in a 27-year-old woman with hereditary folate malabsorption, normal in all respects having completed higher education, who has been treated with parenteral 5-formyltetrahydrofolate since infancy.	Leucovorin	312-336	solute carrier family 46 member 1	Homo sapiens	102-106	
18524888-4	2008	PCFT and RFC produced comparable increases in pemetrexed activity in growth medium with 5-formyltetrahydrofolate.	Leucovorin	88-112	solute carrier family 46 member 1	Homo sapiens	0-4	
18524888-7	2008	PCFT increased the growth inhibitory activity of pemetrexed, but not that of the other antifolates in HepG2 cells grown with 5-formyltetrahydrofolate at physiological pH.	Leucovorin	125-149	solute carrier family 46 member 1	Homo sapiens	0-4