PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33763172-7	2021	On the contrary, overexpression of BECN1 or treating cells with the antioxidant N-acetylcysteine (NAC) could restore the survival of hTERT knockdown cells.	Acetylcysteine	80-96	telomerase reverse transcriptase	Homo sapiens	133-138	
30851366-5	2019	CPT simultaneously increased Nrf2 expression and the level of intracellular reactive oxygen species (ROS), whereas pretreatment with the antioxidants N-acetyl-cysteine (NAC) or glutathione (GSH) strongly attenuated ROS production, which was accompanied by hTERT downregulation.	Acetylcysteine	150-167	telomerase reverse transcriptase	Homo sapiens	256-261	
20363232-9	2010	In addition, NAC blunted the Sp1 reduction and hTERT downregulation by curcumin.	Acetylcysteine	13-16	telomerase reverse transcriptase	Homo sapiens	47-52	
34482039-12	2021	CONCLUSIONS: Reversible alteration of mRNA levels by removing morphine from culture medium, and effect of NAC in co-treatment of morphine plus NAC, emphasize the role of reactive oxygen species in down-regulation of the expression of hTERT and TERF2 by morphine.	Acetylcysteine	106-109	telomerase reverse transcriptase	Homo sapiens	234-239	
34482039-12	2021	CONCLUSIONS: Reversible alteration of mRNA levels by removing morphine from culture medium, and effect of NAC in co-treatment of morphine plus NAC, emphasize the role of reactive oxygen species in down-regulation of the expression of hTERT and TERF2 by morphine.	Acetylcysteine	143-146	telomerase reverse transcriptase	Homo sapiens	234-239	
27654302-7	2017	Meanwhile, fucoidan treatment increased the generation of intracellular ROS, whereas the over-elimination of ROS by N-acetylcysteine, an anti-oxidant, attenuated fucoidan-induced apoptosis, inhibition of hTERT, c-myc, and Sp1 expression, and reversed fucoidan-induced inactivation of the PI3K/Akt signaling pathway.	Acetylcysteine	116-132	telomerase reverse transcriptase	Homo sapiens	204-209	
14963003-9	2004	Likewise, nuclear export of TERT protein, loss in the overall TERT activity, and the onset of replicative senescence were delayed by incubation with N-acetylcysteine.	Acetylcysteine	149-165	telomerase reverse transcriptase	Homo sapiens	28-32	
14963003-9	2004	Likewise, nuclear export of TERT protein, loss in the overall TERT activity, and the onset of replicative senescence were delayed by incubation with N-acetylcysteine.	Acetylcysteine	149-165	telomerase reverse transcriptase	Homo sapiens	62-66	
33763172-7	2021	On the contrary, overexpression of BECN1 or treating cells with the antioxidant N-acetylcysteine (NAC) could restore the survival of hTERT knockdown cells.	Acetylcysteine	98-101	telomerase reverse transcriptase	Homo sapiens	133-138