PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9730105-6	1998	Dogs treated with metoprolol had a marked decrease in CPT-I activity (0.46 +/- 0.03 vs. 0.64 +/- 0.02 micromol min(-1) g(-1) wet weight; P < .02) along with an increase in triglyceride concentration compared to untreated heart failure dogs (3.9 +/- 0.3 v 4.9 +/- 0.2 micromol/g wet weight, respectively; P < .003).	Metoprolol	18-28	carnitine palmitoyltransferase 1B	Rattus norvegicus	54-59	
9730105-8	1998	CONCLUSION: Metoprolol induced a decrease in CPT-I activity and an increase in triglyceride content.	Metoprolol	12-22	carnitine palmitoyltransferase 1B	Rattus norvegicus	45-50	
19233164-4	2009	We therefore hypothesized that metoprolol represses CPT-1B by increasing USF-2-mediated repression of PGC1alpha.	Metoprolol	31-41	carnitine palmitoyltransferase 1B	Rattus norvegicus	52-58	
19233164-9	2009	Chronic metoprolol treatment decreased the expression of CPT-1B in diabetic hearts.	Metoprolol	8-18	carnitine palmitoyltransferase 1B	Rattus norvegicus	57-63	
19233164-11	2009	Metoprolol prevented the increase in PGC1alpha occupation of the CPT-1B promoter region observed in the diabetic heart without affecting PPAR-alpha occupation.	Metoprolol	0-10	carnitine palmitoyltransferase 1B	Rattus norvegicus	65-71