PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31651086-6	2020	RESULTS: We have found that after treated with overexpression of miR-124-3p and MDZ, there exhibited elevated miR-124-3p, declined expression of PIM-1, attenuated migration, invasion, proliferation and colony formation ability, and promoted apoptosis of HepG2 cells.	Midazolam	80-83	microRNA 124-3	Homo sapiens	110-120	
31651086-10	2020	CONCLUSION: Our study found that MDZ could inhibit proliferation and accelerate apoptosis of HCC cells by elevation of miR-124-3p and suppressing PIM-1, which may be an effective method in the treatment of HCC.	Midazolam	33-36	microRNA 124-3	Homo sapiens	119-129