PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32433937-0	2020	Midazolam contributes to neuroprotection against hypoxia/reoxygenation-induced brain injury in neonatal rats via regulation of EAAT2.	Midazolam	0-9	solute carrier family 1 member 2	Rattus norvegicus	127-132	
32433937-4	2020	The present study aimed to detect the role of EAAT2 in the neuroprotective effect of midazolam in neonatal rat brain subjected to H/R.	Midazolam	85-94	solute carrier family 1 member 2	Rattus norvegicus	46-51	
32433937-5	2020	Pretreatment with midazolam reversed H/R-induced apoptosis and downregulation of EAAT2 mRNA and protein expression in the hippocampus.	Midazolam	18-27	solute carrier family 1 member 2	Rattus norvegicus	81-86	
32433937-6	2020	Pretreatment with dihydrokainic acid (a selective inhibitor of EAAT2) exacerbated apoptosis, and thus inhibited the neuroprotective effect of midazolam against H/R injury.	Midazolam	142-151	solute carrier family 1 member 2	Rattus norvegicus	63-68	
32433937-7	2020	We demonstrated for the first time that dysregulation of EAAT2 expression may be related to the neural injury induced by H/R in rat pups, and pretreatment with midazolam attenuated apoptosis and improved learning and memory partly due to regulating EAAT2 expression.	Midazolam	160-169	solute carrier family 1 member 2	Rattus norvegicus	57-62	
32433937-7	2020	We demonstrated for the first time that dysregulation of EAAT2 expression may be related to the neural injury induced by H/R in rat pups, and pretreatment with midazolam attenuated apoptosis and improved learning and memory partly due to regulating EAAT2 expression.	Midazolam	160-169	solute carrier family 1 member 2	Rattus norvegicus	249-254