PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21521783-3	2011	Here we demonstrate using a tamoxifen-inducible Cre-mediated loss of function mouse model that DOT1L, an H3K79 methyltransferase, is required for both initiation and maintenance of MLL-AF9-induced leukemogenesis in vitro and in vivo.	Tamoxifen	28-37	DOT1-like, histone H3 methyltransferase (S. cerevisiae)	Mus musculus	95-100	
30513362-4	2019	METHOD: We used cartilage-specific heterozygous and postnatal tamoxifen-inducible Dot1l knockout mice and performed destabilization of the medial meniscus (DMM) and ageing as OA models.	Tamoxifen	62-71	DOT1-like, histone H3 methyltransferase (S. cerevisiae)	Mus musculus	82-87