PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1850310-5	1991	Epinephrine caused a dose-related reduction of chemiluminescence, superoxide anion generation, enzyme release (lysozyme and beta-glucuronidase), and adhesion to endothelial cell (EC) monolayers in human PMN activated by N-formyl-methionyl-leucyl-phenyl-alanine (fMLP).	Epinephrine	0-11	glucuronidase beta	Homo sapiens	124-142	
26661222-6	2016	The increased uptake of phosphorylated human beta-glucuronidase was inhibited by mannose 6-phosphate for the agents (+-)epinephrine and retinoic acid and by L-NG-nitroarginine methyl ester for the agent lipopolysaccharide in neonatal and adult mice.	Epinephrine	116-131	glucuronidase beta	Homo sapiens	45-63	
5649642-5	1968	Acid phosphatase, beta-glucuronidase and adenylate kinase were released to a small extent during second phase aggregation by ADP or adrenaline; thrombin and collagen particles caused significantly greater release of beta-glucuronidase than of either acid phosphatase or of adenylate kinase.4.	Epinephrine	132-142	glucuronidase beta	Homo sapiens	18-36	
4151956-3	1974	Epinephrine reduced the release of beta-glucuronidase from neutrophils in the presence of zymosan during 2-30 min of incubation and elicited a concomitant elevation of adenosine 3":5"-monophosphate levels.	Epinephrine	0-11	glucuronidase beta	Homo sapiens	35-53	
4364334-7	1974	During contact of neutrophils with RA serum-treated zymosan particles epinephrine, isoproterenol, and cyclic AMP inhibited both particle ingestion and beta-glucuronidase discharge.	Epinephrine	70-81	glucuronidase beta	Homo sapiens	151-169