PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10350488-1	1999	Photoaffinity labeling of the Torpedo nicotinic acetylcholine receptor (nAChR) with [3H]d-tubocurarine (dTC) has identified a residue within the gamma-subunit which, along with the analogous residue in delta-subunit, confers selectivity in binding affinities between the two agonist sites for dTC and alpha-conotoxin (alpha Ctx) MI.	Tubocurarine	88-102	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	38-70	
33326305-5	2021	Of nAChR antagonists tested, D-tubocurarine (D-TC) depressed DRP amplitude the most (43% of control) and actions were restricted to the A-fiber-evoked DRP and selective depression of Adelta-evoked synaptic EFPs (36% of control).	Tubocurarine	29-43	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	3-8	
33326305-5	2021	Of nAChR antagonists tested, D-tubocurarine (D-TC) depressed DRP amplitude the most (43% of control) and actions were restricted to the A-fiber-evoked DRP and selective depression of Adelta-evoked synaptic EFPs (36% of control).	Tubocurarine	45-49	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	3-8	
33326305-9	2021	DC-shifts were produced via nAChRs on primary afferents: they were also seen with nAChR agonists (epibatidine and nicotine), blocked with D-TC but not GABAA receptor blockers, and retained after block of voltage-gated Na+ channels.	Tubocurarine	138-142	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	28-33	
14762152-3	2004	The nicotine response was blocked by d-tubocurarine and mecamylamine but not alpha-bungarotoxin, indicating the presence of calcium permeable, non-alpha7 nicotinic acetylcholine receptor (nAChR) subtype.	Tubocurarine	37-51	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	188-193	
32304995-5	2020	Nicotine-induced PTS acceleration was sensitive to the general nAChR inhibitors mecamylamine and d-tubocurarine as well as to the alpha3beta4-nAChR antagonist alpha-conotoxin AulB, but not to other antagonists primarily addressing alpha3beta2-nAChR or alpha4-, alpha7- and alpha9-containing nAChR.	Tubocurarine	97-111	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	63-68	
19326440-2	2009	Nicotine (classical nAChR agonist) induced cell proliferation, whereas nAChR antagonists, d- tubocurarine or alpha-cobratoxin (alpha-CbT), induced cell death.	Tubocurarine	90-105	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	71-76	
10350488-1	1999	Photoaffinity labeling of the Torpedo nicotinic acetylcholine receptor (nAChR) with [3H]d-tubocurarine (dTC) has identified a residue within the gamma-subunit which, along with the analogous residue in delta-subunit, confers selectivity in binding affinities between the two agonist sites for dTC and alpha-conotoxin (alpha Ctx) MI.	Tubocurarine	88-102	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	72-77