PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18164911-1	2008	The objective of this study was to determine whether cerebrospinal fluid(CSF)-corrected concentrations of N-acetylaspartate are lower in several brain regions of drug- and medication-free subjects with bipolar disorder as compared with matched healthy controls.	N-acetylaspartate	106-123	colony stimulating factor 2	Homo sapiens	73-76	
3116332-2	1987	A high CSF/plasma concentration ratio of N-acetylaspartate indicates that this substance originates in the brain.	N-acetylaspartate	41-58	colony stimulating factor 2	Homo sapiens	7-10	
28334553-1	2017	A multi-matrix hydrophilic interaction liquid chromatography tandem mass spectrometric method (HILIC-MS/MS) was developed for the quantitation of N-Acetyl Aspartic acid (NAA) using stable isotope labeled internal standard, D3-NAA in various biological matrices such as human plasma, human CSF, mouse plasma, brain and spinal cord.	N-acetylaspartate	146-168	colony stimulating factor 2	Homo sapiens	289-292	
28334553-1	2017	A multi-matrix hydrophilic interaction liquid chromatography tandem mass spectrometric method (HILIC-MS/MS) was developed for the quantitation of N-Acetyl Aspartic acid (NAA) using stable isotope labeled internal standard, D3-NAA in various biological matrices such as human plasma, human CSF, mouse plasma, brain and spinal cord.	N-acetylaspartate	170-173	colony stimulating factor 2	Homo sapiens	289-292	
28334553-10	2017	NAA CSF levels in control human subjects (73.3+-31.0ng/mL,N=10) were found to be slightly higher than ALS patients (46.1+-22.6ng/mL, N=10) (P=0.04).	N-acetylaspartate	0-3	colony stimulating factor 2	Homo sapiens	4-7	
21872444-4	2011	CSF-corrected "total NAA" (tNAA; N-acetyl-aspartate + N-acetyl-aspartyl-glutamate), glutamate + glutamine (Glx), creatine + phosphocreatine (Cr + PCr), choline compounds (Cho), and myo-inositol (mI) were assayed in manually drawn regions-of-interest partitioned into gray matter, white matter, and CSF and then coregistered with MRSI.	N-acetylaspartate	21-24	colony stimulating factor 2	Homo sapiens	0-3	
11969317-0	2002	A fully automated method for tissue segmentation and CSF-correction of proton MRSI metabolites corroborates abnormal hippocampal NAA in schizophrenia.	N-acetylaspartate	129-132	colony stimulating factor 2	Homo sapiens	53-56