PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11886524-4 2001 Following sulfur mustard exposure, primary skin fibroblasts from PARP-deficient mice demonstrated increased internucleosomal DNA cleavage, caspase-3 processing and activity, and annexin V positivity, compared to those derived from PARP(+/+) animals. Mustard Gas 10-24 poly (ADP-ribose) polymerase family, member 1 Mus musculus 65-69 11886524-8 2001 In contrast to fibroblasts, keratinocytes from both PARP(-/-) and PARP(+/+) mice express markers of apoptosis in response to sulfur mustard exposure. Mustard Gas 125-139 poly (ADP-ribose) polymerase family, member 1 Mus musculus 52-56 11886524-8 2001 In contrast to fibroblasts, keratinocytes from both PARP(-/-) and PARP(+/+) mice express markers of apoptosis in response to sulfur mustard exposure. Mustard Gas 125-139 poly (ADP-ribose) polymerase family, member 1 Mus musculus 66-70 11886524-9 2001 The effects of PARP on the mode of cell death in different skin cell types may determine the severity of vesication in vivo, and thus have implications for the design of PARP inhibitors to reduce sulfur mustard pathology. Mustard Gas 196-210 poly (ADP-ribose) polymerase family, member 1 Mus musculus 15-19 11886524-9 2001 The effects of PARP on the mode of cell death in different skin cell types may determine the severity of vesication in vivo, and thus have implications for the design of PARP inhibitors to reduce sulfur mustard pathology. Mustard Gas 196-210 poly (ADP-ribose) polymerase family, member 1 Mus musculus 170-174