PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11886524-4	2001	Following sulfur mustard exposure, primary skin fibroblasts from PARP-deficient mice demonstrated increased internucleosomal DNA cleavage, caspase-3 processing and activity, and annexin V positivity, compared to those derived from PARP(+/+) animals.	Mustard Gas	10-24	poly (ADP-ribose) polymerase family, member 1	Mus musculus	65-69	
11886524-8	2001	In contrast to fibroblasts, keratinocytes from both PARP(-/-) and PARP(+/+) mice express markers of apoptosis in response to sulfur mustard exposure.	Mustard Gas	125-139	poly (ADP-ribose) polymerase family, member 1	Mus musculus	52-56	
11886524-8	2001	In contrast to fibroblasts, keratinocytes from both PARP(-/-) and PARP(+/+) mice express markers of apoptosis in response to sulfur mustard exposure.	Mustard Gas	125-139	poly (ADP-ribose) polymerase family, member 1	Mus musculus	66-70	
11886524-9	2001	The effects of PARP on the mode of cell death in different skin cell types may determine the severity of vesication in vivo, and thus have implications for the design of PARP inhibitors to reduce sulfur mustard pathology.	Mustard Gas	196-210	poly (ADP-ribose) polymerase family, member 1	Mus musculus	15-19	
11886524-9	2001	The effects of PARP on the mode of cell death in different skin cell types may determine the severity of vesication in vivo, and thus have implications for the design of PARP inhibitors to reduce sulfur mustard pathology.	Mustard Gas	196-210	poly (ADP-ribose) polymerase family, member 1	Mus musculus	170-174