PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15531761-7	2005	15d-PGJ(2), fluticasone, and salmeterol all inhibited TNFalpha-induced histone H4 acetylation at the eotaxin promoter and NF-kappaB p65 binding to the eotaxin promoter and induced PPARgamma and GR association with the eotaxin promoter, as analyzed by chromatin immunoprecipitation assay.	15d-pgj	0-7	RELA proto-oncogene, NF-kB subunit	Homo sapiens	132-135	
22387200-9	2012	15d-PGJ(2) was observed to induce p65 glutathionylation and is suppressed by a GSH synthesis inhibitor, buthionine sulfoximine, by catalase, and by Nrf2 siRNA molecules.	15d-pgj	0-7	RELA proto-oncogene, NF-kB subunit	Homo sapiens	34-37	
22387200-10	2012	Our results thus indicate that the GSH/ROS-dependent glutathionylation of p65 is likely to be responsible for 15d-PGJ(2)-mediated NF-kappaB inactivation and for the enhanced inhibitory effects of 15d-PGJ(2) on TNFalpha-treated ECs.	15d-pgj	110-117	RELA proto-oncogene, NF-kB subunit	Homo sapiens	74-77	
22387200-10	2012	Our results thus indicate that the GSH/ROS-dependent glutathionylation of p65 is likely to be responsible for 15d-PGJ(2)-mediated NF-kappaB inactivation and for the enhanced inhibitory effects of 15d-PGJ(2) on TNFalpha-treated ECs.	15d-pgj	196-203	RELA proto-oncogene, NF-kB subunit	Homo sapiens	74-77	
16434095-11	2006	NF-kappaB DNA binding activity and NF-kappaB p65 protein expression was inhibited by treatment with 15d-PGJ(2) in human amnion.	15d-pgj	100-107	RELA proto-oncogene, NF-kB subunit	Homo sapiens	45-48