PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16482098-9	2006	By contrast, amlodipine-treated patients showed a higher vascular CTGF, TGF-beta, and collagen IV staining than before treatment.	Amlodipine	13-23	transforming growth factor beta 1	Homo sapiens	72-80	
32246666-9	2020	Conclusion: These preliminary results suggest that TGF-b1 may play a crucial role in the pathogenesis of amlodipine-induced GO.	Amlodipine	105-115	transforming growth factor beta 1	Homo sapiens	51-57	
16221098-4	2005	RESULTS: Amlodipine was found to induce TGF-beta synthesis from mononuclear cells with increasing concentrations, while it was found to inhibit TNF-alpha secretion with increasing concentrations.	Amlodipine	9-19	transforming growth factor beta 1	Homo sapiens	40-48	
16221098-0	2005	TGF-beta and TNF-alpha producing effects of losartan and amlodipine on human mononuclear cell culture.	Amlodipine	57-67	transforming growth factor beta 1	Homo sapiens	0-8	
16221098-6	2005	CONCLUSION: Anti-atherosclerotic effects of amlodipine and losartan might be through increased secretion of TGF-beta from mononuclear cells.	Amlodipine	44-54	transforming growth factor beta 1	Homo sapiens	108-116	
11274244-10	2001	Losartan and amlodipine had opposite effects on TGF-beta(1).	Amlodipine	13-23	transforming growth factor beta 1	Homo sapiens	48-59	
12748343-11	2003	CONCLUSION: Losartan and amlodipine, with similar control of BP, showed different effects on urine protein or TGF-beta1 excretion.	Amlodipine	25-35	transforming growth factor beta 1	Homo sapiens	110-119	
11274244-14	2001	The present study documents that with similar control of BP, losartan and amlodipine have opposite effects on renal hemodynamics and on TGF-beta1 concentrations.	Amlodipine	74-84	transforming growth factor beta 1	Homo sapiens	136-145