PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11343573-4	2000	in desipramine- and citalopram-treated rats was abolished, indicating a desensitization of 5-HT1A receptors.	Desipramine	3-14	5-hydroxytryptamine receptor 1A	Rattus norvegicus	91-97	
8415816-3	1993	Pretreatment with desipramine and mianserin in combination induced the most intense 5-HT syndrome and the greatest fall in colonic temperature after injection of the 5-HT1A agonist 8-hydroxy-2-(di-n-propylamino)-tetralin (8-OH-DPAT).	Desipramine	18-29	5-hydroxytryptamine receptor 1A	Rattus norvegicus	166-172	
9564441-10	1998	The decreases in 5-HT1A mRNA and binding, as well as the MR/GR alterations, were prevented in animals that received imipramine or desipramine antidepressant treatment.	Desipramine	130-141	5-hydroxytryptamine receptor 1A	Rattus norvegicus	17-23	
8415816-0	1993	Chronic, combined treatment with desipramine and mianserin: enhanced 5-HT1A receptor function and altered 5-HT1A/5-HT2 receptor interaction in rats.	Desipramine	33-44	5-hydroxytryptamine receptor 1A	Rattus norvegicus	69-75	
8415816-0	1993	Chronic, combined treatment with desipramine and mianserin: enhanced 5-HT1A receptor function and altered 5-HT1A/5-HT2 receptor interaction in rats.	Desipramine	33-44	5-hydroxytryptamine receptor 1A	Rattus norvegicus	106-112	
1839385-0	1991	Similar effects of treatment with desipramine and electroconvulsive shock on 5-hydroxytryptamine1A receptors in rat brain.	Desipramine	34-45	5-hydroxytryptamine receptor 1A	Rattus norvegicus	77-98	
1839385-1	1991	The effect of chronic and acute treatment with desipramine (DMI) and electroconvulsive shock (ECS) on 5-hydroxytryptamine1A (5-HT1A) receptors was determined in the cortex and the hippocampus brain regions of rats.	Desipramine	47-58	5-hydroxytryptamine receptor 1A	Rattus norvegicus	102-123	
1839385-1	1991	The effect of chronic and acute treatment with desipramine (DMI) and electroconvulsive shock (ECS) on 5-hydroxytryptamine1A (5-HT1A) receptors was determined in the cortex and the hippocampus brain regions of rats.	Desipramine	47-58	5-hydroxytryptamine receptor 1A	Rattus norvegicus	125-131	
1839385-1	1991	The effect of chronic and acute treatment with desipramine (DMI) and electroconvulsive shock (ECS) on 5-hydroxytryptamine1A (5-HT1A) receptors was determined in the cortex and the hippocampus brain regions of rats.	Desipramine	60-63	5-hydroxytryptamine receptor 1A	Rattus norvegicus	102-123	
1839385-1	1991	The effect of chronic and acute treatment with desipramine (DMI) and electroconvulsive shock (ECS) on 5-hydroxytryptamine1A (5-HT1A) receptors was determined in the cortex and the hippocampus brain regions of rats.	Desipramine	60-63	5-hydroxytryptamine receptor 1A	Rattus norvegicus	125-131	
1839385-2	1991	We observed that chronic treatment with both DMI and ECS significantly decreased 5-HT1A receptors, as determined by [3H]8-hydroxy-2-(di-n-propylamino)tetralin [( 3H]8-OH-DPAT) binding, in the cortex but not in the hippocampus.	Desipramine	45-48	5-hydroxytryptamine receptor 1A	Rattus norvegicus	81-87	
1387021-16	1992	These results show that DMI treatment blunted the sensitivity of post-synaptic alpha 2-adrenoceptors, accompanied by complex interactions manifested as increased responsiveness of alpha 1-adrenoceptors and 5-HT1A receptors.	Desipramine	24-27	5-hydroxytryptamine receptor 1A	Rattus norvegicus	180-212