PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32714402-0	2020	Tyrosine Kinase Receptors Axl and MerTK Mediate the Beneficial Effect of Electroacupuncture in a Cuprizone-Induced Demyelinating Model.	Cuprizone	97-106	AXL receptor tyrosine kinase	Mus musculus	26-29	
21569627-4	2011	Axl is upregulated in various disease states, such as in the cuprizone toxicity-induced model of demyelination and in multiple sclerosis (MS) lesions, suggesting that it plays a role in disease pathogenesis.	Cuprizone	61-70	AXL receptor tyrosine kinase	Mus musculus	0-3	
18804096-0	2008	Axl-/- mice have delayed recovery and prolonged axonal damage following cuprizone toxicity.	Cuprizone	72-81	AXL receptor tyrosine kinase	Mus musculus	0-3	
18804096-2	2008	To evaluate Axl"s role in brain during cuprizone toxicity, mice were fed cuprizone and evaluated at 3-, 4-, and 6-week cuprizone treatment and 3- and 5-week post-cuprizone withdrawal.	Cuprizone	39-48	AXL receptor tyrosine kinase	Mus musculus	12-15	
18804096-5	2008	At 6-week cuprizone treatment, there was significantly more Oil Red O+ staining in the Axl-/- corpora callosa than in the WT indicating a lag in the clearance of cellular and myelin debris.	Cuprizone	10-19	AXL receptor tyrosine kinase	Mus musculus	87-90	
18804096-7	2008	Electron microscopy determined that at 3-week post-cuprizone withdrawal the number of dystrophic axons and axons containing autophagosome-like vacuoles/mouse was increased in the Axl-/- mice relative to the WT mice.	Cuprizone	51-60	AXL receptor tyrosine kinase	Mus musculus	179-182	
28925029-0	2017	Loss of Gas6 and Axl signaling results in extensive axonal damage, motor deficits, prolonged neuroinflammation, and less remyelination following cuprizone exposure.	Cuprizone	145-154	AXL receptor tyrosine kinase	Mus musculus	17-20