PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26896736-5	2016	Sal was able to inhibit TGF-beta1-induced EMT phenotypic transition and the activation of key signaling molecules involved in Smad (p-Smad2/3,Snail1) and non-Smad (beta-catenin, p-p38 MAPK) signals which cooperatively regulate the induction of EMT.	salinomycin	0-3	snail family transcriptional repressor 1	Homo sapiens	142-148	
30273566-10	2018	salinomycin treatment attenuated TGF-beta1-induced epithelial-mesenchymal transition (EMT), as evidenced by its ability to increase E-cadherin expression and decrease N-cadherin, Snail and MMP-2 expressions in RCC cells.	salinomycin	0-11	snail family transcriptional repressor 1	Homo sapiens	179-184	
26228105-10	2015	It was also observed that SAL micellar formulations inhibited invasion and harnessed EMT in spite of induced expression of Snail.	salinomycin	26-29	snail family transcriptional repressor 1	Homo sapiens	123-128	
26201092-8	2015	Salinomycin, but not paclitaxel, increased apoptosis, decreased the migration capacity of MCF-7 MS cells, accompanied by a decreased expression of c-Myc, Bcl-2 and Snail, the target genes of the Hh pathway.	salinomycin	0-11	snail family transcriptional repressor 1	Homo sapiens	164-169	
23176396-12	2012	Contrary to expectations, salinomycin induced the expression of EMT markers Snail, vimentin, and Zeb-1, decreased expression of E-cadherin, and also induced phosphorylation of Akt and its downstream targets GSK3-beta and mTOR.	salinomycin	26-37	snail family transcriptional repressor 1	Homo sapiens	76-81