PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21246566-8	2011	RESULTS: The anti-GBM effect of PCH4 is four times more than BP.	butylidenephthalide	61-63	tRNA splicing endonuclease subunit 54	Homo sapiens	32-36	
20809206-2	2012	PCH4, a derivative of n-butylidenephthalide, has been investigated for its anti-tumor effects on oral squamous cell carcinoma (OSCC).	butylidenephthalide	22-43	tRNA splicing endonuclease subunit 54	Homo sapiens	0-4	
21246566-14	2011	CONCLUSIONS: In conclusion, PCH4, a derivative of BP, induced Nur77-mediated apoptosis via the JNK pathway and this mechanism, which is different from that of BP, may explain the increase in the anti-tumor effects on GBM.	butylidenephthalide	50-52	tRNA splicing endonuclease subunit 54	Homo sapiens	28-32	
21246566-14	2011	CONCLUSIONS: In conclusion, PCH4, a derivative of BP, induced Nur77-mediated apoptosis via the JNK pathway and this mechanism, which is different from that of BP, may explain the increase in the anti-tumor effects on GBM.	butylidenephthalide	159-161	tRNA splicing endonuclease subunit 54	Homo sapiens	28-32